Maleate nephrotoxicity: mechanisms of injury and correlates with ischemic/hypoxic tubular cell death.
about
Intracellular oxygen determined by respiration regulates localization of Ras and prenylated proteins.Progressive histone alterations and proinflammatory gene activation: consequences of heme protein/iron-mediated proximal tubule injuryAcute kidney injury: a springboard for progression in chronic kidney diseaseMCP-1 gene activation marks acute kidney injuryRenal cortical albumin gene induction and urinary albumin excretion in response to acute kidney injury.Renal Cortical Lactate Dehydrogenase: A Useful, Accurate, Quantitative Marker of In Vivo Tubular Injury and Acute Renal Failure.Maleic Acid--but Not Structurally Related Methylmalonic Acid--Interrupts Energy Metabolism by Impaired Calcium Homeostasis.HMG-CoA reductase activation and urinary pellet cholesterol elevations in acute kidney injury.PTEN loss defines a TGF-β-induced tubule phenotype of failed differentiation and JNK signaling during renal fibrosis.Marked protection against acute renal and hepatic injury after nitrited myoglobin + tin protoporphyrin administration.Drug-induced Fanconi syndrome associated with fumaric acid esters treatment for psoriasis: a case series.Endotoxin mediates recruitment of RNA polymerase II to target genes in acute renal failure.Combined iron sucrose and protoporphyrin treatment protects against ischemic and toxin-mediated acute renal failureRenal ischemia-induced cholesterol loading: transcription factor recruitment and chromatin remodeling along the HMG CoA reductase gene.Growth and development alter susceptibility to acute renal injury.Renal cortical pyruvate depletion during AKI.Non-clinical toxicological considerations for pharmaceutical salt selection.Progress in the development of animal models of acute kidney injury and its impact on drug discovery.Allopurinol in renal ischemia.Failed Tubule Recovery, AKI-CKD Transition, and Kidney Disease Progression.The role of glycine in regulated cell death.The nephroprotection exerted by curcumin in maleate-induced renal damage is associated with decreased mitochondrial fission and autophagy.Curcumin prevents maleate-induced nephrotoxicity: relation to hemodynamic alterations, oxidative stress, mitochondrial oxygen consumption and activity of respiratory complex I.Elevation in and persistence of multiple urinary biomarkers indicative of oxidative DNA stress and inflammation: Toxicological implications of maleic acid consumption using a rat model.Early detection of renal damage caused by fumaric acid ester therapy by determination of urinary β2-microglobulin.Salt Selection Matters: Differential Renal Toxicity With MDV1634.Maleate Versus MDV1634.2HCl.
P2860
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P2860
Maleate nephrotoxicity: mechanisms of injury and correlates with ischemic/hypoxic tubular cell death.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
Maleate nephrotoxicity: mechan ...... ic/hypoxic tubular cell death.
@en
Maleate nephrotoxicity: mechan ...... ic/hypoxic tubular cell death.
@nl
type
label
Maleate nephrotoxicity: mechan ...... ic/hypoxic tubular cell death.
@en
Maleate nephrotoxicity: mechan ...... ic/hypoxic tubular cell death.
@nl
prefLabel
Maleate nephrotoxicity: mechan ...... ic/hypoxic tubular cell death.
@en
Maleate nephrotoxicity: mechan ...... ic/hypoxic tubular cell death.
@nl
P2093
P2860
P1476
Maleate nephrotoxicity: mechan ...... ic/hypoxic tubular cell death.
@en
P2093
Ali C M Johnson
Karol Bomsztyk
Masayo Naito
Richard A Zager
P2860
P304
P356
10.1152/AJPRENAL.00434.2007
P577
2007-10-17T00:00:00Z