Endoplasmic reticulum stress is sufficient for the induction of IL-1β production via activation of the NF-κB and inflammasome pathways. - Wikidata - wikimedia - TriplyDB

Endoplasmic reticulum stress is sufficient for the induction of IL-1β production via activation of the NF-κB and inflammasome pathways.

Endoplasmic reticulum stress is sufficient for the induction of IL-1β production via activation of the NF-κB and inflammasome pathways.

http://www.wikidata.org/entity/Q46972930

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Interplay between Inflammation and Cellular Stress Triggered by Flaviviridae Viruses Adipokines and the role of visceral adipose tissue in inflammatory bowel disease Natural Compounds as Regulators of NLRP3 Inflammasome-Mediated IL-1β Production Initiation and perpetuation of NLRP3 inflammasome activation and assembly Silver nanoparticles induce degradation of the endoplasmic reticulum stress sensor activating transcription factor-6 leading to activation of the NLRP-3 inflammasome Inflammasome activation in response to the Yersinia type III secretion system requires hyperinjection of translocon proteins YopB and YopD.Toll-like receptor 4 promotes autonomic dysfunction, inflammation and microglia activation in the hypothalamic paraventricular nucleus: role of endoplasmic reticulum stress.Endoplasmic reticulum stress is increased in adipose tissue of women with gestational diabetes.Endoplasmic reticulum stress-induced IRE1α activation mediates cross-talk of GSK-3β and XBP-1 to regulate inflammatory cytokine production.Synergistic Effects of Cilostazol and Probucol on ER Stress-Induced Hepatic Steatosis via Heme Oxygenase-1-Dependent Activation of Mitochondrial Biogenesis.Dietary saturated fatty acid and polyunsaturated fatty acid oppositely affect hepatic NOD-like receptor protein 3 inflammasome through regulating nuclear factor-kappa B activation Involvement of endoplasmic reticulum stress in angiotensin II-induced NLRP3 inflammasome activation in human renal proximal tubular cells in vitro Targeting heme oxygenase-1 and carbon monoxide for therapeutic modulation of inflammation.White adipose tissue cells and the progression of cachexia: inflammatory pathways.Hepatoprotective Effect of Quercetin on Endoplasmic Reticulum Stress and Inflammation after Intense Exercise in Mice through Phosphoinositide 3-Kinase and Nuclear Factor-Kappa B.The evolving paradigm of cell-nonautonomous UPR-based regulation of immunity by cancer cells.The Role of Damage-Associated Molecular Patterns (DAMPs) in Human Diseases: Part II: DAMPs as diagnostics, prognostics and therapeutics in clinical medicine.Innate immunity in diabetes and diabetic nephropathy.The entangled ER-mitochondrial axis as a potential therapeutic strategy in neurodegeneration: A tangled duo unchained.Transplantation and Damage-Associated Molecular Patterns (DAMPs).Guanylate Binding Proteins regulate inflammasome activation in response to hyper-injected Yersinia translocon components.Anti-inflammatory actions of (-)-epicatechin in the adipose tissue of obese mice.The Role of Endoplasmic Reticulum Stress in Cardiovascular Disease and Exercise.Aberrant DNA-PKcs and ERGIC1 expression may be involved in initiation of gastric cancer.Carbon Monoxide in Lung Cell Physiology and Disease.Melatonin as an Anti-Inflammatory Agent Modulating Inflammasome Activation.The level of physical training modulates cytokine levels through P2X7 receptor in healthy subjects.Coordinating Organismal Metabolism During Protein Misfolding in the ER Through the Unfolded Protein Response.Integrated Immunomodulatory Mechanisms through which Long-Chain n-3 Polyunsaturated Fatty Acids Attenuate Obese Adipose Tissue Dysfunction.Melatonin attenuated early brain injury induced by subarachnoid hemorrhage via regulating NLRP3 inflammasome and apoptosis signaling.NF-κB Blockade in Hypothalamic Paraventricular Nucleus Inhibits High-Salt-Induced Hypertension Through NLRP3 and Caspase-1.Differential Regulation of Toll-Like Receptor-Mediated Cytokine Production by Unfolded Protein Response.p58 Is an Endogenous Neuroprotectant for Retinal Ganglion Cells

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Endoplasmic reticulum stress is sufficient for the induction of IL-1β production via activation of the NF-κB and inflammasome pathways.

http://www.wikidata.org/entity/Q46972930

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

@zh-my

2013年学术文章

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2013年學術文章

@yue

2013年學術文章

@zh-hant

name

Endoplasmic reticulum stress i ...... -κB and inflammasome pathways.

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Endoplasmic reticulum stress i ...... -κB and inflammasome pathways.

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type

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Endoplasmic reticulum stress i ...... -κB and inflammasome pathways.

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Endoplasmic reticulum stress i ...... -κB and inflammasome pathways.

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Endoplasmic reticulum stress i ...... -κB and inflammasome pathways.

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Endoplasmic reticulum stress i ...... -κB and inflammasome pathways.

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Innate Immunity

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Endoplasmic reticulum stress i ...... F-κB and inflammasome pathways

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Hun Taeg Chung

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Min Zheng

http://www.w3.org/2001/XMLSchema#string

Sang Won Park

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Stefan W Ryter

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Sun Oh Jeong

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Sung Hoon Back

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Yeonsoo Joe

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P2860

Mammalian transcription factor ATF6 is synthesized as a transmembrane protein and activated by proteolysis in response to endoplasmic reticulum stress

The inflammasomes

Role of SERCA1 truncated isoform in the proapoptotic calcium transfer from ER to mitochondria during ER stress

Seed Oil of Brucea javanica Induces Apoptotic Death of Acute Myeloid Leukemia Cells via Both the Death Receptors and the Mitochondrial-Related Pathways

Interleukin-1 in the pathogenesis and treatment of inflammatory diseases

Innate immune activation through Nalp3 inflammasome sensing of asbestos and silica

Oxidative protein folding in eukaryotes: mechanisms and consequences

Signal integration in the endoplasmic reticulum unfolded protein response

A role for mitochondria in NLRP3 inflammasome activation

Identification of thioredoxin-binding protein-2/vitamin D(3) up-regulated protein 1 as a negative regulator of thioredoxin function and expression

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endoplasmic reticulum