Netrin-1 Promotes Inflammation Resolution to Achieve Endothelialization of Small-Diameter Tissue Engineering Blood Vessels by Improving Endothelial Progenitor Cells Function In Situ.

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The Host Response in Tissue Engineering: Crosstalk Between Immune cells and Cell-laden Scaffolds

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Netrin-1 Promotes Inflammation Resolution to Achieve Endothelialization of Small-Diameter Tissue Engineering Blood Vessels by Improving Endothelial Progenitor Cells Function In Situ.

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2017 nî lūn-bûn

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2017年学术文章

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Netrin-1 Promotes Inflammation ...... enitor Cells Function In Situ.

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Netrin-1 Promotes Inflammation ...... enitor Cells Function In Situ.

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Netrin-1 Promotes Inflammation ...... enitor Cells Function In Situ.

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Netrin-1 Promotes Inflammation ...... enitor Cells Function In Situ.

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Netrin-1 Promotes Inflammation ...... enitor Cells Function In Situ.

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Netrin-1 Promotes Inflammation ...... enitor Cells Function In Situ.

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P1476

Netrin-1 Promotes Inflammation ...... enitor Cells Function In Situ.

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P2093

Chuhong Zhu

http://www.w3.org/2001/XMLSchema#string

Da Huo

http://www.w3.org/2001/XMLSchema#string

Feila Liu

http://www.w3.org/2001/XMLSchema#string

Gang Li

http://www.w3.org/2001/XMLSchema#string

Ge Guan

http://www.w3.org/2001/XMLSchema#string

Ge Liu

http://www.w3.org/2001/XMLSchema#string

Mingcan Yang

http://www.w3.org/2001/XMLSchema#string

Ning Ding

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Simin Wan

http://www.w3.org/2001/XMLSchema#string

Wang Cai

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Interleukin-10 deficiency impairs bone marrow-derived endothelial progenitor cell survival and function in ischemic myocardium

A cautionary tale for autologous vascular tissue engineering: impact of human demographics on the ability of adipose-derived mesenchymal stem cells to recruit and differentiate into smooth muscle cells.

Membrane vesicles as conveyors of immune responses

Tissue-engineered vascular grafts transform into mature blood vessels via an inflammation-mediated process of vascular remodeling.

The use of BDNF to enhance the patency rate of small-diameter tissue-engineered blood vessels through stem cell homing mechanisms.

Noncoding RNAs and atherosclerosis

Inflammation 2010: new adventures of an old flame.

Netrin-1 induces angiogenesis via a DCC-dependent ERK1/2-eNOS feed-forward mechanism

Tissue engineering therapy for cardiovascular disease.

Aspirin-induced histone acetylation in endothelial cells enhances synthesis of the secreted isoform of netrin-1 thus inhibiting monocyte vascular infiltration.

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1700278

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scholarly article

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10.1002/ADVS.201700278

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2017-09-28T00:00:00Z

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29270340

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tissue engineering

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5738088

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Netrin-1 Promotes Inflammation Resolution to Achieve Endothelialization of Small-Diameter Tissue Engineering Blood Vessels by Improving Endothelial Progenitor Cells Function In Situ.

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Netrin-1 Promotes Inflammation Resolution to Achieve Endothelialization of Small-Diameter Tissue Engineering Blood Vessels by Improving Endothelial Progenitor Cells Function In Situ.

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