Disruption of nNOS-NOS1AP protein-protein interactions suppresses neuropathic pain in mice.

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ZLc002, a putative small-molecule inhibitor of nNOS interaction with NOS1AP, suppresses inflammatory nociception and chemotherapy-induced neuropathic pain and synergizes with paclitaxel to reduce tumor cell viability

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Q56890049-CBF9AF42-5A45-4030-B72A-94661FD29853

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Disruption of nNOS-NOS1AP protein-protein interactions suppresses neuropathic pain in mice.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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Disruption of nNOS-NOS1AP prot ...... sses neuropathic pain in mice.

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Disruption of nNOS-NOS1AP prot ...... sses neuropathic pain in mice.

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Disruption of nNOS-NOS1AP prot ...... sses neuropathic pain in mice.

@en

Disruption of nNOS-NOS1AP prot ...... sses neuropathic pain in mice.

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Disruption of nNOS-NOS1AP prot ...... sses neuropathic pain in mice.

@en

Disruption of nNOS-NOS1AP prot ...... sses neuropathic pain in mice.

@nl

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Disruption of nNOS-NOS1AP prot ...... sses neuropathic pain in mice.

@en

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Aarti Chawla

http://www.w3.org/2001/XMLSchema#string

Andrea G Hohmann

http://www.w3.org/2001/XMLSchema#string

Andy Hudmon

http://www.w3.org/2001/XMLSchema#string

Wan-Hung Lee

http://www.w3.org/2001/XMLSchema#string

Yvonne Y Lai

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Specific coupling of NMDA receptor activation to nitric oxide neurotoxicity by PSD-95 protein

Independent human MAP-kinase signal transduction pathways defined by MEK and MKK isoforms

CAPON: a protein associated with neuronal nitric oxide synthase that regulates its interactions with PSD95

MKK3- and MKK6-regulated gene expression is mediated by the p38 mitogen-activated protein kinase signal transduction pathway

Sciatic nerve ligation-induced proliferation of spinal cord astrocytes is mediated by kappa opioid activation of p38 mitogen-activated protein kinase

Mechanisms involved in the development of chemotherapy-induced neuropathy

Solution structure of the extended neuronal nitric oxide synthase PDZ domain complexed with an associated peptide

The nNOS-p38MAPK pathway is mediated by NOS1AP during neuronal death

MAP kinase and pain.

Formation of nNOS/PSD-95 PDZ dimer requires a preformed beta-finger structure from the nNOS PDZ domain.

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scholarly article

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10.1097/J.PAIN.0000000000001152

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English

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Li-Li Li

Michael J Courtney

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2018-01-09T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

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29319606

http://www.w3.org/2001/XMLSchema#string

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neuropathic pain

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6085107

http://www.w3.org/2001/XMLSchema#string

Disruption of nNOS-NOS1AP protein-protein interactions suppresses neuropathic pain in mice.

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P2860

P2860

Disruption of nNOS-NOS1AP protein-protein interactions suppresses neuropathic pain in mice.

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P1433

P1476

P2093

P2860

P31

P356

P407

P50

P577

P698

P921

P932

P356

P1433

P1476

P2093

P2860

P31

P356

P407

P50

P577

P698

P921

P932