RNA interference in Histoplasma capsulatum demonstrates a role for alpha-(1,3)-glucan in virulence.
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Surface architecture of histoplasma capsulatumHost pathogen relations: exploring animal models for fungal pathogensAlternative oxidase mediates pathogen resistance in Paracoccidioides brasiliensis infectionExpression of Paracoccidioides brasiliensis AMY1 in a Histoplasma capsulatum amy1 mutant, relates an α-(1,4)-amylase to cell wall α-(1,3)-glucan synthesisBiochemical characterization of Paracoccidioides brasiliensis α-1,3-glucanase Agn1p, and its functionality by heterologous Expression in Schizosaccharomyces pombeTranscriptome Profile of the Response of Paracoccidioides spp. to a Camphene Thiosemicarbazide DerivativeThe spectrum of fungi that infects humansStructural features responsible for the biological stability of Histoplasma's virulence factor CBP.Reverse genetics through random mutagenesis in Histoplasma capsulatum.Cell organisation, sulphur metabolism and ion transport-related genes are differentially expressed in Paracoccidioides brasiliensis mycelium and yeast cells.The transcriptome analysis of early morphogenesis in Paracoccidioides brasiliensis mycelium reveals novel and induced genes potentially associated to the dimorphic processHemoglobin uptake by Paracoccidioides spp. is receptor-mediatedParacoccidioides spp. catalases and their role in antioxidant defense against host defense responsesGenetically altering the expression of neutral trehalase gene affects conidiospore thermotolerance of the entomopathogenic fungus Metarhizium acridum.Discordant influence of Blastomyces dermatitidis yeast-phase-specific gene BYS1 on morphogenesis and virulenceNeurospora crassa 1,3-α-glucan synthase, AGS-1, is required for cell wall biosynthesis during macroconidia development.Insights into Fungal Morphogenesis and Immune Evasion: Fungal conidia, when situated in mammalian lungs, may switch from mold to pathogenic yeasts or spore-forming spherules.Conidia but not yeast cells of the fungal pathogen Histoplasma capsulatum trigger a type I interferon innate immune response in murine macrophages.Comparison of phylogenetically distinct Histoplasma strains reveals evolutionarily divergent virulence strategiesGlycosylation and immunoreactivity of the Histoplasma capsulatum Cfp4 yeast-phase exoantigenA 32-kilodalton hydrolase plays an important role in Paracoccidioides brasiliensis adherence to host cells and influences pathogenicity.Lipidomic analysis of extracellular vesicles from the pathogenic phase of Paracoccidioides brasiliensis.Comparative transcriptomics of the saprobic and parasitic growth phases in Coccidioides spp.Agglutination of Histoplasma capsulatum by IgG monoclonal antibodies against Hsp60 impacts macrophage effector functionsGenome update of the dimorphic human pathogenic fungi causing paracoccidioidomycosis.Identification of a copper-inducible promoter for use in ectopic expression in the fungal pathogen Histoplasma capsulatumDefinition of the extracellular proteome of pathogenic-phase Histoplasma capsulatumInhibition of PbGP43 expression may suggest that gp43 is a virulence factor in Paracoccidioides brasiliensis.A Paracoccidioides brasiliensis glycan shares serologic and functional properties with cryptococcal glucuronoxylomannan.P. brasiliensis virulence is affected by SconC, the negative regulator of inorganic sulfur assimilation.Histoplasma yeast and mycelial transcriptomes reveal pathogenic-phase and lineage-specific gene expression profiles.The transcriptional repressor TupA in Aspergillus niger is involved in controlling gene expression related to cell wall biosynthesis, development, and nitrogen source availability.Efficient implementation of RNA interference in the pathogenic yeast Cryptococcus neoformansCoevolution of morphology and virulence in Candida species.RNA interference in fungi: pathways, functions, and applications.Histoplasma mechanisms of pathogenesis--one portfolio doesn't fit all.Chronological aging is associated with biophysical and chemical changes in the capsule of Cryptococcus neoformansHistoplasma capsulatum alpha-(1,3)-glucan blocks innate immune recognition by the beta-glucan receptor.Immunization with apoptotic phagocytes containing Histoplasma capsulatum activates functional CD8(+) T cells to protect against histoplasmosisLoss of cell wall alpha(1-3) glucan affects Cryptococcus neoformans from ultrastructure to virulence.
P2860
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P2860
RNA interference in Histoplasma capsulatum demonstrates a role for alpha-(1,3)-glucan in virulence.
description
2004 nî lūn-bûn
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2004年の論文
@ja
2004年学术文章
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2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
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name
RNA interference in Histoplasm ...... pha-(1,3)-glucan in virulence.
@en
RNA interference in Histoplasm ...... pha-(1,3)-glucan in virulence.
@nl
type
label
RNA interference in Histoplasm ...... pha-(1,3)-glucan in virulence.
@en
RNA interference in Histoplasm ...... pha-(1,3)-glucan in virulence.
@nl
prefLabel
RNA interference in Histoplasm ...... pha-(1,3)-glucan in virulence.
@en
RNA interference in Histoplasm ...... pha-(1,3)-glucan in virulence.
@nl
P2860
P1476
RNA interference in Histoplasm ...... pha-(1,3)-glucan in virulence.
@en
P2093
Jacquelyn T Engle
P2860
P304
P356
10.1111/J.1365-2958.2004.04131.X
P407
P577
2004-07-01T00:00:00Z