CCR2+ inflammatory monocytes are recruited to Yersinia pyogranulomas and dictate adaptive responses at the expense of innate immunity during oral infection.

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All Are Not Created Equal: Phenotypic Adaptation to Distinct Niches Within Mammalian Tissues

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CCR2+ inflammatory monocytes are recruited to Yersinia pyogranulomas and dictate adaptive responses at the expense of innate immunity during oral infection.

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2017 nî lūn-bûn

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2017年学术文章

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CCR2+ inflammatory monocytes a ...... mmunity during oral infection.

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CCR2+ inflammatory monocytes a ...... mmunity during oral infection.

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CCR2+ inflammatory monocytes a ...... mmunity during oral infection.

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CCR2+ inflammatory monocytes a ...... mmunity during oral infection.

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CCR2+ inflammatory monocytes a ...... mmunity during oral infection.

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CCR2+ inflammatory monocytes a ...... mmunity during oral infection.

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CCR2+ inflammatory monocytes a ...... mmunity during oral infection.

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Adrianus W M van der Velden

http://www.w3.org/2001/XMLSchema#string

Brian S Sheridan

http://www.w3.org/2001/XMLSchema#string

Yue Zhang

http://www.w3.org/2001/XMLSchema#string

P2860

In situ imaging of the endogenous CD8 T cell response to infection.

The effects of inflammatory cytokines on lymphatic endothelial barrier function

CD8(+) T cells restrict Yersinia pseudotuberculosis infection: bypass of anti-phagocytosis by targeting antigen-presenting cells

TNFα and IFNγ but not perforin are critical for CD8 T cell-mediated protection against pulmonary Yersinia pestis infection.

Oral infection drives a distinct population of intestinal resident memory CD8(+) T cells with enhanced protective function

Local Inflammatory Cues Regulate Differentiation and Persistence of CD8+ Tissue-Resident Memory T Cells

YopJ-promoted cytotoxicity and systemic colonization are associated with high levels of murine interleukin-18, gamma interferon, and neutrophils in a live vaccine model of Yersinia pseudotuberculosis infection

Effector CD8+ T cells are generated in response to an immunodominant epitope in type III effector YopE during primary Yersinia pseudotuberculosis infection

ROS-inhibitory activity of YopE is required for full virulence of Yersinia in mice

The presence of professional phagocytes dictates the number of host cells targeted for Yop translocation during infection

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scholarly article

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10.1128/IAI.00782-17

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English

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James Bliska

Camille Khairallah

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2017-12-20T00:00:00Z

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29263104

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P932

5820931

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CCR2+ inflammatory monocytes are recruited to Yersinia pyogranulomas and dictate adaptive responses at the expense of innate immunity during oral infection.

about

P2860

P2860

CCR2+ inflammatory monocytes are recruited to Yersinia pyogranulomas and dictate adaptive responses at the expense of innate immunity during oral infection.

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P31

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