Molecular mechanisms and physiological relevance of RGK proteins in the heart.

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Molecular mechanisms and physiological relevance of RGK proteins in the heart.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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Molecular mechanisms and physiological relevance of RGK proteins in the heart.

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Molecular mechanisms and physiological relevance of RGK proteins in the heart.

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Molecular mechanisms and physiological relevance of RGK proteins in the heart.

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Molecular mechanisms and physiological relevance of RGK proteins in the heart.

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Molecular mechanisms and physiological relevance of RGK proteins in the heart.

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Molecular mechanisms and physiological relevance of RGK proteins in the heart.

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P1476

Molecular mechanisms and physiological relevance of RGK proteins in the heart.

@en

P2093

D Beqollari

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R A Bannister

http://www.w3.org/2001/XMLSchema#string

U Meza

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P2860

The deltaC isoform of CaMKII is activated in cardiac hypertrophy and induces dilated cardiomyopathy and heart failure

14-3-3 and calmodulin control subcellular distribution of Kir/Gem and its regulation of cell shape and calcium channel activity

Rad GTPase deficiency leads to cardiac hypertrophy

Phosphorylation of critical serine residues in Gem separates cytoskeletal reorganization from down-regulation of calcium channel activity

Dominant negative suppression of Rad leads to QT prolongation and causes ventricular arrhythmias via modulation of L-type Ca2+ channels in the heart

Loss of Ca(v)1.3 (CACNA1D) function in a human channelopathy with bradycardia and congenital deafness

Gem: an induced, immediate early protein belonging to the Ras family

Rad: a member of the Ras family overexpressed in muscle of type II diabetic humans

Regulation of voltage-gated calcium channel activity by the Rem and Rad GTPases

The GTP binding proteins Gem and Rad are negative regulators of the Rho-Rho kinase pathway

P31

scholarly article

review article

P356

10.1111/APHA.13016

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P577

2017-12-13T00:00:00Z

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P698

29237245

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