Pretreatment with intraventricular basic fibroblast growth factor decreases infarct size following focal cerebral ischemia in rats.
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Synergistic protective effect of caspase inhibitors and bFGF against brain injury induced by transient focal ischaemiaThe changing landscape of ischaemic brain injury mechanisms.Experience-associated structural events, subependymal cellular proliferative activity, and functional recovery after injury to the central nervous system.Differential cellular FGF-2 upregulation in the rat facial nucleus following axotomy, functional electrical stimulation and corticosterone: a possible therapeutic target to Bell's palsy.Angiogenic factors in the central nervous system.Neuroprotective action of cycloheximide involves induction of bcl-2 and antioxidant pathwaysIntracisternal basic fibroblast growth factor enhances functional recovery and up-regulates the expression of a molecular marker of neuronal sprouting following focal cerebral infarctionThe choroid plexus as a site of damage in hemorrhagic and ischemic stroke and its role in responding to injury.SUN11602, a novel aniline compound, mimics the neuroprotective mechanisms of basic fibroblast growth factor.FGF-2 immunoreactivity in adult rat ependyma and choroid plexus: responses to global forebrain ischemia and intraventricular FGF-2.Postischemic intraventricular administration of FGF-2 expressing adenoviral vectors improves neurologic outcome and reduces infarct volume after transient focal cerebral ischemia in rats.Adenoviral gene transfer of basic fibroblast growth factor promotes angiogenesis in rat brain.Transient and widespread astroglial activation in the brain after a striatal 6-OHDA-induced partial lesion of the nigrostriatal system.Delayed treatment with intravenous basic fibroblast growth factor reduces infarct size following permanent focal cerebral ischemia in rats.Increased vulnerability of NFH-LacZ transgenic mouse to traumatic brain injury-induced behavioral deficits and cortical damage.The gene for heparin-binding epidermal growth factor-like growth factor is stress-inducible: its role in cerebral ischemia.Evidence that brain-derived neurotrophic factor neuroprotection is linked to its ability to reverse the NMDA-induced inactivation of protein kinase C in cortical neurons.Human FGF-1 gene delivery protects against quinolinate-induced striatal and hippocampal injury in neonatal rats.Permeability of injured blood brain barrier for exogenous bFGF and protection mechanism of bFGF in rat brain ischemia.
P2860
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P2860
Pretreatment with intraventricular basic fibroblast growth factor decreases infarct size following focal cerebral ischemia in rats.
description
1994 nî lūn-bûn
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1994年の論文
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1994年学术文章
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1994年学术文章
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1994年学术文章
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1994年学术文章
@zh-hans
1994年学术文章
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1994年學術文章
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name
Pretreatment with intraventric ...... cal cerebral ischemia in rats.
@en
Pretreatment with intraventric ...... cal cerebral ischemia in rats.
@nl
type
label
Pretreatment with intraventric ...... cal cerebral ischemia in rats.
@en
Pretreatment with intraventric ...... cal cerebral ischemia in rats.
@nl
prefLabel
Pretreatment with intraventric ...... cal cerebral ischemia in rats.
@en
Pretreatment with intraventric ...... cal cerebral ischemia in rats.
@nl
P2093
P356
P1433
P1476
Pretreatment with intraventric ...... cal cerebral ischemia in rats.
@en
P2093
Berlove DJ
Finklestein SP
Moskowitz MA
P2860
P304
P356
10.1002/ANA.410350413
P577
1994-04-01T00:00:00Z