Insulinoma-associated 1: A novel nuclear marker in Merkel cell carcinoma (cutaneous neuroendocrine carcinoma).

about

Insulinoma-associated 1: A novel nuclear marker in Merkel cell carcinoma (cutaneous neuroendocrine carcinoma).

Item

http://www.wikidata.org/entity/Q47392838

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Insulinoma-associated 1: A nov ...... ous neuroendocrine carcinoma).

@en

Insulinoma-associated 1: A novel nuclear marker in Merkel cell carcinoma

@nl

type

Item

label

Insulinoma-associated 1: A nov ...... ous neuroendocrine carcinoma).

@en

Insulinoma-associated 1: A novel nuclear marker in Merkel cell carcinoma

@nl

prefLabel

Insulinoma-associated 1: A nov ...... ous neuroendocrine carcinoma).

@en

Insulinoma-associated 1: A novel nuclear marker in Merkel cell carcinoma

@nl

P1476

Insulinoma-associated 1: A nov ...... ous neuroendocrine carcinoma).

@en

P2093

Daniel D Bennett

http://www.w3.org/2001/XMLSchema#string

Madhuchhanda Roy

http://www.w3.org/2001/XMLSchema#string

Rebecca M Baus

http://www.w3.org/2001/XMLSchema#string

Ricardo V Lloyd

http://www.w3.org/2001/XMLSchema#string

P2860

Neuroendocrine differentiation factor, IA-1, is a transcriptional repressor and contains a specific DNA-binding domain: identification of consensus IA-1 binding sequence

IA1 is NGN3-dependent and essential for differentiation of the endocrine pancreas.

INSM1 functions as a transcriptional repressor of the neuroD/beta2 gene through the recruitment of cyclin D1 and histone deacetylases

Zinc finger transcription factor INSM1 interrupts cyclin D1 and CDK4 binding and induces cell cycle arrest

Identification of an INSM1-binding site in the insulin promoter: negative regulation of the insulin gene transcription

NeuroD1/E47 regulates the E-box element of a novel zinc finger transcription factor, IA-1, in developing nervous system.

Insm1 promotes endocrine cell differentiation by modulating the expression of a network of genes that includes Neurog3 and Ripply3

Insm1 controls development of pituitary endocrine cells and requires a SNAG domain for function and for recruitment of histone-modifying factors

Insm1 (IA-1) is a crucial component of the transcriptional network that controls differentiation of the sympatho-adrenal lineage

Structure, expression, and biological function of INSM1 transcription factor in neuroendocrine differentiation.

P304

129-135

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1111/CUP.13079

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Jason N Rosenbaum

Patrick S Rush

P577

2017-11-17T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29148079

http://www.w3.org/2001/XMLSchema#string