Bystander destruction of hematopoietic progenitor and stem cells in a mouse model of infusion-induced bone marrow failure.
about
Current concepts in the pathophysiology and treatment of aplastic anemiaThe complex and central role of interferon-γ in graft-versus-host disease and graft-versus-tumor activityImmunological Basis of Bone Marrow Failure after Allogeneic Hematopoietic Stem Cell TransplantationAnimal models for acquired bone marrow failure syndromes.Post-hematopoietic stem cell transplantion immune-mediated cytopenias.The role of the Th1 transcription factor T-bet in a mouse model of immune-mediated bone-marrow failureEzh2 regulates transcriptional and posttranslational expression of T-bet and promotes Th1 cell responses mediating aplastic anemia in mice.Th17 immune responses contribute to the pathophysiology of aplastic anemia.MHC class II upregulation and colocalization with Fas in experimental models of immune-mediated bone marrow failureImmune-mediated bone marrow failure in C57BL/6 micePromotion of Expansion and Differentiation of Hematopoietic Stem Cells by Interleukin-27 into Myeloid Progenitors to Control Infection in Emergency Myelopoiesis.IFN-γ-mediated hematopoietic cell destruction in murine models of immune-mediated bone marrow failure.Lymphocytes with aberrant expression of Fas or Fas ligand attenuate immune bone marrow failure in a mouse modelHydrogen therapy may be an effective and specific novel treatment for aplastic anemia.Allogeneic Th1 cells home to host bone marrow and spleen and mediate IFNγ-dependent aplasia.Retrovirus-specificity of regulatory T cells is neither present nor required in preventing retrovirus-induced bone marrow immune pathology.Role of perforin-mediated cell apoptosis in murine models of infusion-induced bone marrow failure.Hematopoietic stem cell development, aging and functional failure.New therapeutic approaches for protecting hematopoietic stem cells in aplastic anemia.Concise review: the bone marrow niche as a target of graft versus host disease.T-cell-restricted T-bet overexpression induces aberrant hematopoiesis of myeloid cells and impairs function of macrophages in the lung.New challenges to developing animal models for human immune-mediated marrow failure.Experimental bone marrow failure in mice ameliorated by OCH via tippling the balance of released cytokines from Th1 to Th2.Hematopoietic stem cell loss and hematopoietic failure in severe aplastic anemia is driven by macrophages and aberrant podoplanin expression
P2860
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P2860
Bystander destruction of hematopoietic progenitor and stem cells in a mouse model of infusion-induced bone marrow failure.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
@zh-cn
2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年學術文章
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2004年學術文章
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name
Bystander destruction of hemat ...... n-induced bone marrow failure.
@en
Bystander destruction of hemat ...... n-induced bone marrow failure.
@nl
type
label
Bystander destruction of hemat ...... n-induced bone marrow failure.
@en
Bystander destruction of hemat ...... n-induced bone marrow failure.
@nl
prefLabel
Bystander destruction of hemat ...... n-induced bone marrow failure.
@en
Bystander destruction of hemat ...... n-induced bone marrow failure.
@nl
P2093
P1433
P1476
Bystander destruction of hemat ...... on-induced bone marrow failure
@en
P2093
Felicia M Ellison
Jichun Chen
Karen Lipovsky
Neal S Young
P304
P356
10.1182/BLOOD-2004-03-1115
P407
P577
2004-05-27T00:00:00Z