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Evolutionary History of the Smyd Gene Family in Metazoans: A Framework to Identify the Orthologs of Human Smyd Genes in Drosophila and Other Animal SpeciesMissense mutations in the SH3TC2 protein causing Charcot-Marie-Tooth disease type 4C affect its localization in the plasma membrane and endocytic pathway.Peptides encoded by short ORFs control development and define a new eukaryotic gene familyControl of Distal-less expression in the Drosophila appendages by functional 3' enhancers.Planar cell polarity controls directional Notch signaling in the Drosophila leg.The Drosophila junctophilin gene is functionally equivalent to its four mammalian counterparts and is a modifier of a Huntingtin poly-Q expansion and the Notch pathway.Mitochondrial defects and neuromuscular degeneration caused by altered expression of Drosophila Gdap1: implications for the Charcot-Marie-Tooth neuropathy.Dynamic EGFR-Ras signalling in Drosophila leg development.
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description
hulumtues
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researcher
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wetenschapper
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հետազոտող
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name
Máximo Ibo Galindo
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Máximo Ibo Galindo
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Máximo Ibo Galindo
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Máximo Ibo Galindo
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Máximo Ibo Galindo
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type
label
Máximo Ibo Galindo
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Máximo Ibo Galindo
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Máximo Ibo Galindo
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Máximo Ibo Galindo
@nl
Máximo Ibo Galindo
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prefLabel
Máximo Ibo Galindo
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Máximo Ibo Galindo
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Máximo Ibo Galindo
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Máximo Ibo Galindo
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Máximo Ibo Galindo
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P1053
A-5299-2010
P106
P1153
7006443930
P21
P31
P3829
P496
0000-0001-8448-9760