about
Selective extra-dimensional set shifting deficit in a knock-in mouse model of Huntington's diseaseRobust MR-based approaches to quantifying white matter structure and structure/function alterations in Huntington's disease.Neuroimaging Biomarkers Predict Brain Structural Connectivity Change in a Mouse Model of Vascular Cognitive Impairment.A Critical Re-Examination of the Intraluminal Filament MCAO Model: Impact of External Carotid Artery Transection.In Vivo MRI Evidence that Neuropathology is Attenuated by Cognitive Enrichment in the Yac128 Huntington's Disease Mouse Model.Longitudinal in vivo MRI in a Huntington's disease mouse model: Global atrophy in the absence of white matter microstructural damage.Intra-arterial nitroglycerin as directed acute treatment in experimental ischemic stroke.Operant-based instrumental learning for analysis of genetically modified models of Huntington's disease.Infections Up to 76 Days After Stroke Increase Disability and Death.Rule learning, visuospatial function and motor performance in the Hdh(Q92) knock-in mouse model of Huntington's disease.The IMPROVE Guidelines (Ischaemia Models: Procedural Refinements Of in Vivo Experiments).Behavioral outcome measures to improve experimental stroke research.Lickometry: A novel and sensitive method for assessing functional deficits in rats after stroke.Systematic and detailed analysis of behavioural tests in the rat middle cerebral artery occlusion model of stroke: Tests for long-term assessment.Time course of choice reaction time deficits in the Hdh(Q92) knock-in mouse model of Huntington's disease in the operant serial implicit learning task (SILT).Early onset deficits on the delayed alternation task in the Hdh(Q92) knock-in mouse model of Huntington's disease.Assessment of Motor Coordination and Balance in Mice Using the Rotarod, Elevated Bridge, and Footprint Tests.Striatal lesions in the mouse disrupt acquisition and retention, but not implicit learning, in the SILT procedural motor learning task.Small vessels, dementia and chronic diseases - molecular mechanisms and pathophysiology.CDK12 inhibition reduces abnormalities in cells from patients with myotonic dystrophy and in a mouse modelOperant Analysis of Fronto-striatal Function in Rodents
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description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Rebecca C Trueman
@ast
Rebecca C Trueman
@en
Rebecca C Trueman
@es
Rebecca C Trueman
@nl
Rebecca C Trueman
@sl
type
label
Rebecca C Trueman
@ast
Rebecca C Trueman
@en
Rebecca C Trueman
@es
Rebecca C Trueman
@nl
Rebecca C Trueman
@sl
prefLabel
Rebecca C Trueman
@ast
Rebecca C Trueman
@en
Rebecca C Trueman
@es
Rebecca C Trueman
@nl
Rebecca C Trueman
@sl
P106
P21
P31
P496
0000-0002-6746-0912