CD40L mediated alternative NFκB-signaling induces resistance to BCR-inhibitors in patients with mantle cell lymphoma.

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CD40L mediated alternative NFκB-signaling induces resistance to BCR-inhibitors in patients with mantle cell lymphoma.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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CD40L mediated alternative NFκ ...... nts with mantle cell lymphoma.

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CD40L mediated alternative NFκ ...... nts with mantle cell lymphoma.

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label

CD40L mediated alternative NFκ ...... nts with mantle cell lymphoma.

@en

CD40L mediated alternative NFκ ...... nts with mantle cell lymphoma.

@nl

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CD40L mediated alternative NFκ ...... nts with mantle cell lymphoma.

@en

CD40L mediated alternative NFκ ...... nts with mantle cell lymphoma.

@nl

P1476

CD40L mediated alternative NFκ ...... nts with mantle cell lymphoma.

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Andreas Rosenwald

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Hilka Rauert-Wunderlich

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Ingolf Berberich

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Martina Rudelius

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P2860

The Bruton tyrosine kinase inhibitor PCI-32765 blocks B-cell activation and is efficacious in models of autoimmune disease and B-cell malignancy

Targeting the B-cell receptor signaling pathway in B lymphoid malignancies

Second-generation inhibitors of Bruton tyrosine kinase

Modulation of life and death by the TNF receptor superfamily

Postibrutinib outcomes in patients with mantle cell lymphoma.

Targeting BTK with ibrutinib in relapsed or refractory mantle-cell lymphoma

Cyclin D1 overexpression in non-Hodgkin's lymphoma with chromosome 11 bcl-1 rearrangement.

Bruton tyrosine kinase inhibitor ibrutinib (PCI-32765) has significant activity in patients with relapsed/refractory B-cell malignancies.

Cell-cycle reprogramming for PI3K inhibition overrides a relapse-specific C481S BTK mutation revealed by longitudinal functional genomics in mantle cell lymphoma

The IKK inhibitor Bay 11-7082 induces cell death independent from inhibition of activation of NFκB transcription factors.

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scholarly article

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10.1038/S41419-017-0157-6

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P433

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P478

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P577

2018-01-24T00:00:00Z

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29367645

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P932

5833745

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