GPR120 is not required for ω-3 PUFAs-induced cell growth inhibition and apoptosis in breast cancer cells.

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GPR120 is not required for ω-3 PUFAs-induced cell growth inhibition and apoptosis in breast cancer cells.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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2017年學術文章

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GPR120 is not required for ω-3 ...... ptosis in breast cancer cells.

@en

GPR120 is not required for ω-3 ...... ptosis in breast cancer cells.

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Item

label

GPR120 is not required for ω-3 ...... ptosis in breast cancer cells.

@en

GPR120 is not required for ω-3 ...... ptosis in breast cancer cells.

@nl

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GPR120 is not required for ω-3 ...... ptosis in breast cancer cells.

@en

GPR120 is not required for ω-3 ...... ptosis in breast cancer cells.

@nl

P1476

GPR120 is not required for ω-3 ...... optosis in breast cancer cells

@en

P2093

Guangxiao Lin

http://www.w3.org/2001/XMLSchema#string

Jianping Gong

http://www.w3.org/2001/XMLSchema#string

Shenglong Zhu

http://www.w3.org/2001/XMLSchema#string

Siyuan Jiang

http://www.w3.org/2001/XMLSchema#string

Wei Chen

http://www.w3.org/2001/XMLSchema#string

Xiaowei Jiang

http://www.w3.org/2001/XMLSchema#string

Yong Q Chen

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P2860

GPR120 is an omega-3 fatty acid receptor mediating potent anti-inflammatory and insulin-sensitizing effects

Oleate promotes the proliferation of breast cancer cells via the G protein-coupled receptor GPR40

ω-3 free fatty acids and all-trans retinoic acid synergistically induce growth inhibition of three subtypes of breast cancer cell lines

Distribution and regulation of protein expression of the free fatty acid receptor GPR120.

Omega-3 fatty acids reduce obesity-induced tumor progression independent of GPR120 in a mouse model of postmenopausal breast cancer.

Omega-3 DHA- and EPA-dopamine conjugates induce PPARγ-dependent breast cancer cell death through autophagy and apoptosis.

Selective small-molecule agonists of G protein-coupled receptor 40 promote glucose-dependent insulin secretion and reduce blood glucose in mice

Dietary PUFA and cancer.

Inflammation increases NOTCH1 activity via MMP9 and is counteracted by Eicosapentaenoic Acid-free fatty acid in colon cancer cells.

FGF21 treatment ameliorates alcoholic fatty liver through activation of AMPK-SIRT1 pathway.

P304

180-186

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P31

scholarly article

P356

10.1002/CBIN.10883

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Zhao He

P577

2017-12-15T00:00:00Z

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P698

28980737

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