Mitochondrial Impairment as a Key Factor for the Lack of Attachment after Cold Storage of Hepatocyte Suspensions.

about

Mitochondrial Impairment as a Key Factor for the Lack of Attachment after Cold Storage of Hepatocyte Suspensions.

Item

http://www.wikidata.org/entity/Q47640202

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Mitochondrial Impairment as a ...... age of Hepatocyte Suspensions.

@en

Mitochondrial Impairment as a ...... age of Hepatocyte Suspensions.

@nl

type

Item

label

Mitochondrial Impairment as a ...... age of Hepatocyte Suspensions.

@en

Mitochondrial Impairment as a ...... age of Hepatocyte Suspensions.

@nl

prefLabel

Mitochondrial Impairment as a ...... age of Hepatocyte Suspensions.

@en

Mitochondrial Impairment as a ...... age of Hepatocyte Suspensions.

@nl

P1476

Mitochondrial Impairment as a ...... age of Hepatocyte Suspensions.

@en

P2093

Anja Bienholz

http://www.w3.org/2001/XMLSchema#string

Björn Walter

http://www.w3.org/2001/XMLSchema#string

Gesine Pless-Petig

http://www.w3.org/2001/XMLSchema#string

Ursula Rauen

http://www.w3.org/2001/XMLSchema#string

P2860

Treatment of the Crigler-Najjar syndrome type I with hepatocyte transplantation

Cold storage of rat hepatocyte suspensions for one week in a customized cold storage solution--preservation of cell attachment and metabolism

Cold-induced apoptosis of rat liver endothelial cells: contribution of mitochondrial alterations.

Hypothermia injury/cold-induced apoptosis--evidence of an increase in chelatable iron causing oxidative injury in spite of low O2-/H2O2 formation.

Cold-induced apoptosis of hepatocytes: mitochondrial permeability transition triggered by nonmitochondrial chelatable iron.

Hypothermic maintenance of hepatocyte spheroids.

Improvement of the cold storage of blood vessels with a vascular preservation solution. Study in porcine aortic segments.

Survival and function of isolated hepatocytes after cryopreservation.

Mitochondrial dysfunction in the pathogenesis of necrotic and apoptotic cell death.

Hepatocyte viability and adenosine triphosphate content decrease linearly over time during conventional cold storage of rat liver grafts.

P304

1855-1867

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1177/0963689717743254

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-12-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29390882

http://www.w3.org/2001/XMLSchema#string

P932

5802638

http://www.w3.org/2001/XMLSchema#string