Knockout of l-Histidine Decarboxylase Prevents Cholangiocyte Damage and Hepatic Fibrosis in Mice Subjected to High-Fat Diet Feeding via Disrupted Histamine/Leptin Signaling.

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Knockout of l-Histidine Decarboxylase Prevents Cholangiocyte Damage and Hepatic Fibrosis in Mice Subjected to High-Fat Diet Feeding via Disrupted Histamine/Leptin Signaling.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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name

Knockout of l-Histidine Decarb ...... ed Histamine/Leptin Signaling.

@en

Knockout of l-Histidine Decarb ...... ed Histamine/Leptin Signaling.

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type

Item

label

Knockout of l-Histidine Decarb ...... ed Histamine/Leptin Signaling.

@en

Knockout of l-Histidine Decarb ...... ed Histamine/Leptin Signaling.

@nl

prefLabel

Knockout of l-Histidine Decarb ...... ed Histamine/Leptin Signaling.

@en

Knockout of l-Histidine Decarb ...... ed Histamine/Leptin Signaling.

@nl

P1476

Knockout of l-Histidine Decarb ...... ted Histamine/Leptin Signaling

@en

P2093

Fanyin Meng

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Gianfranco Alpini

http://www.w3.org/2001/XMLSchema#string

Heather Francis

http://www.w3.org/2001/XMLSchema#string

Jennifer Demieville

http://www.w3.org/2001/XMLSchema#string

Jennifer M Bailey

http://www.w3.org/2001/XMLSchema#string

Kishore Polireddy

http://www.w3.org/2001/XMLSchema#string

Laura Hargrove

http://www.w3.org/2001/XMLSchema#string

Lindsey Kennedy

http://www.w3.org/2001/XMLSchema#string

Lindsey Stockton

http://www.w3.org/2001/XMLSchema#string

Moises I Nevah Rubin

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P2860

Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor

Hepatic stellate cells: protean, multifunctional, and enigmatic cells of the liver

Progression of non-alcoholic steatosis to steatohepatitis and fibrosis parallels cumulative accumulation of danger signals that promote inflammation and liver tumors in a high fat-cholesterol-sugar diet model in mice.

Cellular senescence in livers from children with end stage liver disease

Gene polymorphisms of cellular senescence marker p21 and disease progression in non-alcohol-related fatty liver disease

Four faces of cellular senescence.

Histamine regulation in glucose and lipid metabolism via histamine receptors: model for nonalcoholic steatohepatitis in mice

Cholangiocyte senescence by way of N-ras activation is a characteristic of primary sclerosing cholangitis

Histopathology of nonalcoholic fatty liver disease/nonalcoholic steatohepatitis

Inactivation of Tm6sf2, a Gene Defective in Fatty Liver Disease, Impairs Lipidation but Not Secretion of Very Low Density Lipoproteins.

P304

600-615

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scholarly article

P356

10.1016/J.AJPATH.2017.11.016

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P407

English

P433

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P478

188

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P50

Amelia Sybenga

P577

2017-12-15T00:00:00Z

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P698

29248461

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P932

5840487

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