Elevated circulating TGF-β is not the cause of increased atherosclerosis development in biglycan deficient mice.

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Elevated circulating TGF-β is not the cause of increased atherosclerosis development in biglycan deficient mice.

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2017 nî lūn-bûn

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Elevated circulating TGF-β is ...... nt in biglycan deficient mice.

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Elevated circulating TGF-β is ...... nt in biglycan deficient mice.

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label

Elevated circulating TGF-β is ...... nt in biglycan deficient mice.

@en

Elevated circulating TGF-β is ...... nt in biglycan deficient mice.

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Elevated circulating TGF-β is ...... nt in biglycan deficient mice.

@en

Elevated circulating TGF-β is ...... nt in biglycan deficient mice.

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P1476

Elevated circulating TGF-β is ...... nt in biglycan deficient mice.

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Adrian K Wyllie

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Alex P Wyllie

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Joel C Thompson

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Lisa R Tannock

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Patricia G Wilson

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P2860

The response-to-retention hypothesis of early atherogenesis

TGF-beta1: a novel target for cardiovascular pharmacology

A brief elevation of serum amyloid A is sufficient to increase atherosclerosis

Distribution of glycosaminoglycans in the intima of human aortas: changes in atherosclerosis and diabetes mellitus

Negative regulation of transforming growth factor-beta by the proteoglycan decorin

Increased atherosclerosis in mice with increased vascular biglycan content.

Biglycan-triggered TLR-2- and TLR-4-signaling exacerbates the pathophysiology of ischemic acute kidney injury.

Biglycan deficiency: increased aortic aneurysm formation and lack of atheroprotection.

Reversibility of renal injury with cholesterol lowering in hyperlipidemic diabetic mice

Renal accumulation of biglycan and lipid retention accelerates diabetic nephropathy.

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68-75

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scholarly article

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10.1016/J.ATHEROSCLEROSIS.2017.11.005

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268

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2017-11-10T00:00:00Z

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29182988

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P921

atherosclerosis

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5750111

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