about
Jumping species-a mechanism for coronavirus persistence and survival.A mouse model for Betacoronavirus subgroup 2c using a bat coronavirus strain HKU5 variantPathogenic influenza viruses and coronaviruses utilize similar and contrasting approaches to control interferon-stimulated gene responses.A comprehensive collection of systems biology data characterizing the host response to viral infection.Allelic Variation in the Toll-Like Receptor Adaptor Protein Ticam2 Contributes to SARS-Coronavirus Pathogenesis in Mice.Interferon regulatory factor 3-dependent pathways are critical for control of herpes simplex virus type 1 central nervous system infection.Coronavirus non-structural protein 16: evasion, attenuation, and possible treatments.Cytokine systems approach demonstrates differences in innate and pro-inflammatory host responses between genetically distinct MERS-CoV isolates.New Metrics for Evaluating Viral Respiratory Pathogenesis.Genome Wide Identification of SARS-CoV Susceptibility Loci Using the Collaborative Cross.Corneal replication is an interferon response-independent bottleneck for virulence of herpes simplex virus 1 in the absence of virion host shutoff.A SARS-like cluster of circulating bat coronaviruses shows potential for human emergence.SARS-like WIV1-CoV poised for human emergence.Release of severe acute respiratory syndrome coronavirus nuclear import block enhances host transcription in human lung cells.Cell host response to infection with novel human coronavirus EMC predicts potential antivirals and important differences with SARS coronavirusMechanisms of severe acute respiratory syndrome coronavirus-induced acute lung injury.Bugs in the system.Reverse genetics with a full-length infectious cDNA of the Middle East respiratory syndrome coronavirus.Control of herpes simplex virus replication is mediated through an interferon regulatory factor 3-dependent pathway.Evaluation of serologic and antigenic relationships between middle eastern respiratory syndrome coronavirus and other coronaviruses to develop vaccine platforms for the rapid response to emerging coronaviruses.Epitope Addition and Ablation via Manipulation of a Dengue Virus Serotype 1 Infectious CloneAttenuation and restoration of severe acute respiratory syndrome coronavirus mutant lacking 2'-o-methyltransferase activity.Herpes simplex virus virion host shutoff attenuates establishment of the antiviral state.MERS-CoV Accessory ORFs Play Key Role for Infection and Pathogenesis.Broad-spectrum antiviral GS-5734 inhibits both epidemic and zoonotic coronaviruses.MERS Vaccine Candidate Offers Promise, but Questions Remain.CD-loop extension in Zika virus envelope protein key for stability and pathogenesis.Viral metagenomics, protein structure, and reverse genetics: Key strategies for investigating coronaviruses.MERS-CoV and H5N1 influenza virus antagonize antigen presentation by altering the epigenetic landscape.Development of a Broadly Accessible Venezuelan Equine Encephalitis Virus Replicon Particle Vaccine Platform.CC002/Unc females are mouse models of exercise-induced paradoxical fat response.Combination Attenuation Offers Strategy for Live Attenuated Coronavirus VaccinesErratum: Corrigendum: A SARS-like cluster of circulating bat coronaviruses shows potential for human emergenceSARS-Like Coronavirus WIV1-CoV Does Not Replicate in Egyptian Fruit Bats ()Middle East Respiratory Syndrome Vaccine Candidates: Cautious OptimismShortening of Zika virus CD-loop reduces neurovirulence while preserving antigenicityReturn of the Coronavirus: 2019-nCoVIsolation and characterization of SARS-CoV-2 from the first US COVID-19 patientSARS-CoV-2 is sensitive to type I interferon pretreatmentTrypsin Treatment Unlocks Barrier for Zoonotic Bat Coronavirus Infection
P50
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P50
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Vineet D Menachery
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Vineet D Menachery
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Vineet D. Menachery
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Vineet D. Menachery
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Vineet D. Menachery
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Vineet D Menachery
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Vineet D Menachery
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Vineet D. Menachery
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Vineet D. Menachery
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Vineet D. Menachery
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Vineet D. Menachery
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Vineet D. Menachery
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Vineet D. Menachery
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Vineet D. Menachery
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P1412
P21
P31
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0000-0001-8803-7606
P569
2000-01-01T00:00:00Z