Extra-placental expression of vascular endothelial growth factor receptor-1, (Flt-1) and soluble Flt-1 (sFlt-1), by peripheral blood mononuclear cells (PBMCs) in normotensive and preeclamptic pregnant women.
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Effect of antihypertensive therapy with alpha methyldopa on levels of angiogenic factors in pregnancies with hypertensive disordersTissue factor and its natural inhibitor in pre-eclampsia and SGAPreeclampsia, hypoxia, thrombosis, and inflammationHypertension and maternal-fetal conflict during placental malariaFully Bayesian tests of neutrality using genealogical summary statistics.Pre-eclampsia part 1: current understanding of its pathophysiology.Maternal circulating levels of activin A, inhibin A, sFlt-1 and endoglin at parturition in normal pregnancy and pre-eclampsia.Expression levels of seven candidate genes in human peripheral blood mononuclear cells and their association with preeclampsia.Do anti-angiogenic or angiogenic factors contribute to the protection of birth weight at high altitude afforded by Andean ancestry?Altered matrix metalloproteinase-2 and -9 expression/activity links placental ischemia and anti-angiogenic sFlt-1 to uteroplacental and vascular remodeling and collagen deposition in hypertensive pregnancy.Synthesis of sFlt-1 by platelet-monocyte aggregates contributes to the pathogenesis of preeclampsia.Role of biomarkers in early detection of preeclampsiaMaternal and fetoplacental hypoxia do not alter circulating angiogenic growth effectors during human pregnancy.Beyond oxygen: complex regulation and activity of hypoxia inducible factors in pregnancy.Mid-pregnancy levels of angiogenic markers as indicators of pathways to preterm delivery.Ouabain inhibits placental sFlt1 production by repressing HSP27-dependent HIF-1α pathway.Unexplained fetal death is associated with increased concentrations of anti-angiogenic factors in amniotic fluidIntegrating multiple 'omics' analyses identifies serological protein biomarkers for preeclampsia.Design of growth factor sequestering biomaterialsReduced uterine perfusion pressure induces hypertension in the pregnant mouse.Circulating angiogenic and antiangiogenic factors in women with eclampsia.Peptidomic Identification of Serum Peptides Diagnosing PreeclampsiaMidpregnancy levels of angiogenic markers in relation to maternal characteristics.Expression of the sFLT1 gene in cord blood cells is associated to maternal arsenic exposure and decreased birth weight.Preeclampsia, a disease of the maternal endothelium: the role of antiangiogenic factors and implications for later cardiovascular disease.Regulation of soluble fms-like tyrosine kinase-1 production in response to placental ischemia/hypoxia: role of angiotensin II.Transcriptionally active syncytial aggregates in the maternal circulation may contribute to circulating soluble fms-like tyrosine kinase 1 in preeclampsia.Relationships among maternal nutrient intake and placental biomarkers during the 1st trimester in low-income women.Microbial invasion of the amniotic cavity in preeclampsia as assessed by cultivation and sequence-based methodsL-arginine supplementation abolishes the blood pressure and endothelin response to chronic increases in plasma sFlt-1 in pregnant rats.Cardiac angiogenic imbalance leads to peripartum cardiomyopathy.Nicotine inhibits cytokine production by placenta cells via NFkappaB: potential role in pregnancy-induced hypertension.Emerging role of relaxin in the maternal adaptations to normal pregnancy: implications for preeclampsiaLate-onset preeclampsia is associated with an imbalance of angiogenic and anti-angiogenic factors in patients with and without placental lesions consistent with maternal underperfusion.Serum and plasma determination of angiogenic and anti-angiogenic factors yield different results: the need for standardization in clinical practice.A prospective cohort study of the value of maternal plasma concentrations of angiogenic and anti-angiogenic factors in early pregnancy and midtrimester in the identification of patients destined to develop preeclampsiaComplement activation induces dysregulation of angiogenic factors and causes fetal rejection and growth restriction.Acute pyelonephritis during pregnancy changes the balance of angiogenic and anti-angiogenic factors in maternal plasma.Preeclampsia and pregnancies with small-for-gestational age neonates have different profiles of complement split products.Agonistic autoantibodies to the angiotensin II type I receptor cause pathophysiologic characteristics of preeclampsia.
P2860
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P2860
Extra-placental expression of vascular endothelial growth factor receptor-1, (Flt-1) and soluble Flt-1 (sFlt-1), by peripheral blood mononuclear cells (PBMCs) in normotensive and preeclamptic pregnant women.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Extra-placental expression of ...... d preeclamptic pregnant women.
@en
Extra-placental expression of ...... d preeclamptic pregnant women.
@nl
type
label
Extra-placental expression of ...... d preeclamptic pregnant women.
@en
Extra-placental expression of ...... d preeclamptic pregnant women.
@nl
prefLabel
Extra-placental expression of ...... d preeclamptic pregnant women.
@en
Extra-placental expression of ...... d preeclamptic pregnant women.
@nl
P2093
P1433
P1476
Extra-placental expression of ...... d preeclamptic pregnant women.
@en
P2093
A Rajakumar
H M Michael
N Markovic
P A Rajakumar
R Thadhani
P304
P356
10.1016/J.PLACENTA.2004.09.001
P577
2005-08-01T00:00:00Z