Increased susceptibility to tumor necrosis factor-alpha in butyric acid-induced apoptosis is caused by downregulation of cFLIP expression in Jurkat T cells.

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Butyric acid induces apoptosis via oxidative stress in Jurkat T-cells.

P2860

Q43075329-CF8F2A79-527D-436A-A08E-98B28E9A93BE

P2860

Increased susceptibility to tumor necrosis factor-alpha in butyric acid-induced apoptosis is caused by downregulation of cFLIP expression in Jurkat T cells.

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http://www.wikidata.org/entity/Q47780857

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2008 nî lūn-bûn

@nan

2008年の論文

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2008年学术文章

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2008年学术文章

@zh

2008年学术文章

@zh-cn

2008年学术文章

@zh-hans

2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

@zh-hant

name

Increased susceptibility to tu ...... expression in Jurkat T cells.

@en

Increased susceptibility to tu ...... expression in Jurkat T cells.

@nl

type

Item

label

Increased susceptibility to tu ...... expression in Jurkat T cells.

@en

Increased susceptibility to tu ...... expression in Jurkat T cells.

@nl

prefLabel

Increased susceptibility to tu ...... expression in Jurkat T cells.

@en

Increased susceptibility to tu ...... expression in Jurkat T cells.

@nl

P1476

Increased susceptibility to tu ...... expression in Jurkat T cells.

@en

P2093

Kuniyasu Ochiai

http://www.w3.org/2001/XMLSchema#string

Shintaro Seto

http://www.w3.org/2001/XMLSchema#string

Tomoko Kurita-Ochiai

http://www.w3.org/2001/XMLSchema#string

P2860

Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes

Inhibition of death receptor signals by cellular FLIP

Cotreatment with histone deacetylase inhibitor LAQ824 enhances Apo-2L/tumor necrosis factor-related apoptosis inducing ligand-induced death inducing signaling complex activity and apoptosis of human acute leukemia cells

Dominant-negative FADD inhibits TNFR60-, Fas/Apo1- and TRAIL-R/Apo2-mediated cell death but not gene induction

Yama/CPP32 beta, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase

The histone deacetylase inhibitor and chemotherapeutic agent suberoylanilide hydroxamic acid (SAHA) induces a cell-death pathway characterized by cleavage of Bid and production of reactive oxygen species

Butyric acid-induced T-cell apoptosis is mediated by caspase-8 and -9 activation in a Fas-independent manner

Biochemical pathways of caspase activation during apoptosis.

Initiator caspases in apoptosis signaling pathways.

The contribution of interleukin-1 and tumor necrosis factor to periodontal tissue destruction.

P304

188-196

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P31

scholarly article

P356

10.1111/J.1348-0421.2008.00023.X

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P433

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P478

http://www.w3.org/2001/XMLSchema#string

P577

2008-03-01T00:00:00Z

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P5875

5449309

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P698

18402601

http://www.w3.org/2001/XMLSchema#string

Increased susceptibility to tumor necrosis factor-alpha in butyric acid-induced apoptosis is caused by downregulation of cFLIP expression in Jurkat T cells.

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P2860

P2860

Increased susceptibility to tumor necrosis factor-alpha in butyric acid-induced apoptosis is caused by downregulation of cFLIP expression in Jurkat T cells.

description

name

type

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P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698