From inflammation to sickness and depression: when the immune system subjugates the brainThe type 1 TNF receptor and its associated adapter protein, FAN, are required for TNFalpha-induced sickness behavior.Luteolin reduces IL-6 production in microglia by inhibiting JNK phosphorylation and activation of AP-1Twenty years of research on cytokine-induced sickness behaviorAlcoholism and inflammation: neuroimmunology of behavioral and mood disordersTen years of Nature Reviews Neuroscience: insights from the highly citedInduction of IDO by bacille Calmette-Guérin is responsible for development of murine depressive-like behaviorCa2+/calmodulin-dependent kinase kinase alpha is expressed by monocytic cells and regulates the activation profile.Voluntary wheel running reverses age-induced changes in hippocampal gene expressionEffects of insulin-like growth factor-I on cytokine-induced sickness behavior in mice.In vitro and in vivo evidence for a role of the P2X7 receptor in the release of IL-1 beta in the murine brainInoculation of Bacillus Calmette-Guerin to mice induces an acute episode of sickness behavior followed by chronic depressive-like behavior.Central administration of lipopolysaccharide induces depressive-like behavior in vivo and activates brain indoleamine 2,3 dioxygenase in murine organotypic hippocampal slice culturesFractalkine receptor (CX3CR1) deficiency sensitizes mice to the behavioral changes induced by lipopolysaccharideCentral administration of insulin-like growth factor-I decreases depressive-like behavior and brain cytokine expression in mice.Lipopolysaccharide-induced depressive-like behavior is mediated by indoleamine 2,3-dioxygenase activation in mice.Microglial/macrophage GRK2 determines duration of peripheral IL-1beta-induced hyperalgesia: contribution of spinal cord CX3CR1, p38 and IL-1 signaling.Intracerebroventricular administration of HIV-1 Tat induces brain cytokine and indoleamine 2,3-dioxygenase expression: a possible mechanism for AIDS comorbid depression.Protein hormones and immunityA global view of twenty years of Brain, Behavior, and ImmunityLipopolysaccharide induces delayed FosB/DeltaFosB immunostaining within the mouse extended amygdala, hippocampus and hypothalamus, that parallel the expression of depressive-like behavior.G protein-coupled receptor kinase 6 acts as a critical regulator of cytokine-induced hyperalgesia by promoting phosphatidylinositol 3-kinase and inhibiting p38 signaling.Novel activity of an anti-inflammatory cytokine: IL-10 prevents TNFalpha-induced resistance to IGF-I in myoblasts.Indoleamine 2,3-dioxygenase inhibition attenuates lipopolysaccharide induced persistent microglial activation and depressive-like complications in fractalkine receptor (CX(3)CR1)-deficient mice.Aging sensitizes mice to behavioral deficits induced by central HIV-1 gp120Methylphenidate prevents high-fat diet (HFD)-induced learning/memory impairment in juvenile miceInterleukin-1 beta converting enzyme is necessary for development of depression-like behavior following intracerebroventricular administration of lipopolysaccharide to mice.Spatio-temporal differences in the profile of murine brain expression of proinflammatory cytokines and indoleamine 2,3-dioxygenase in response to peripheral lipopolysaccharide administration.NMDA receptor blockade by ketamine abrogates lipopolysaccharide-induced depressive-like behavior in C57BL/6J mice.Uncoupling of interleukin-6 from its signalling pathway by dietary n-3-polyunsaturated fatty acid deprivation alters sickness behaviour in mice.Voluntary wheel running does not affect lipopolysaccharide-induced depressive-like behavior in young adult and aged mice.Getting nervous about immunity.Low nociceptor GRK2 prolongs prostaglandin E2 hyperalgesia via biased cAMP signaling to Epac/Rap1, protein kinase Cepsilon, and MEK/ERK.Mannan oligosaccharide modulates gene expression profile in pigs experimentally infected with porcine reproductive and respiratory syndrome virus.Proinflammatory cytokine impairment of insulin-like growth factor I-induced protein synthesis in skeletal muscle myoblasts requires ceramide.Advancing the understanding of behaviors associated with Bacille Calmette Guérin infection using multivariate analysis.Aging exacerbates depressive-like behavior in mice in response to activation of the peripheral innate immune system.Exaggerated expression of skeletal muscle-derived interleukin-6, but not TNFalpha, in mice lacking interleukin-10.Bacille Calmette-Guérin inoculation induces chronic activation of peripheral and brain indoleamine 2,3-dioxygenase in mice.Tumor necrosis factor alpha inhibits cyclin A expression and retinoblastoma hyperphosphorylation triggered by insulin-like growth factor-I induction of new E2F-1 synthesis.
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P50
description
American immunophysiologist, researcher and academic
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հետազոտող
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name
Keith W. Kelley
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Keith W. Kelley
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Keith W. Kelley
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Keith W. Kelley
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Keith W. Kelley
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Keith W. Kelley
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Keith W. Kelley
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Keith W. Kelley
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Keith W. Kelley
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Keith W. Kelley
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Keith W. Kelley
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Keith W. Kelley
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Keith W. Kelley
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Keith W. Kelley
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Keith W. Kelley
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0000-0002-6837-8793
P569
1947-11-05T00:00:00Z