Loss of cytochrome c oxidase promotes RAS-dependent ROS production from the ER resident NADPH oxidase, Yno1p, in yeast.
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Mitochondrial genetic variants identified to be associated with BMI in adultsCalcium and ROS: A mutual interplayProgrammed Cell Death Initiation and Execution in Budding YeastNew insights into the role of mitochondrial dynamics and autophagy during oxidative stress and aging in the heartOma1 Links Mitochondrial Protein Quality Control and TOR Signaling To Modulate Physiological Plasticity and Cellular Stress Responses.Mitochondrial Metabolism in Aging HeartTranscriptome analysis illuminates the nature of the intracellular interaction in a vertebrate-algal symbiosis.Overexpression of UCP1 in tobacco induces mitochondrial biogenesis and amplifies a broad stress response.The progression of cell death affects the rejection of allogeneic tumors in immune-competent mice - implications for cancer therapy.De novo comparative transcriptome analysis of Acremonium chrysogenum: high-yield and wild-type strains of cephalosporin C producer.Mitochondrial GWA Analysis of Lipid Profile Identifies Genetic Variants to Be Associated with HDL Cholesterol and Triglyceride Levels.Cardiac deficiency of single cytochrome oxidase assembly factor scox induces p53-dependent apoptosis in a Drosophila cardiomyopathy modelResveratrol ameliorates mitochondrial dysfunction but increases the risk of hypoglycemia following hemorrhagic shock.Oxidative stress in fungi: its function in signal transduction, interaction with plant hosts, and lignocellulose degradationImproved transcription and translation with L-leucine stimulation of mTORC1 in Roberts syndromeBdf1 Bromodomains Are Essential for Meiosis and the Expression of Meiotic-Specific Genes.Mitochondrion to endoplasmic reticulum apposition length in zebrafish embryo spinal progenitors is unchanged in response to perturbations associated with Alzheimer's diseaseHomocysteine injures vascular endothelial cells by inhibiting mitochondrial activity.The control of translational accuracy is a determinant of healthy ageing in yeast.Cellular redox homeostasis, reactive oxygen species and replicative ageing in Saccharomyces cerevisiae.The biochemistry and cell biology of aging: metabolic regulation through mitochondrial signaling.The oxidation state of the cytoplasmic glutathione redox system does not correlate with replicative lifespan in yeastStarvation signals in yeast are integrated to coordinate metabolic reprogramming and stress response to ensure longevity.Tristetraprolin inhibits mitochondrial function through suppression of α-Synuclein expression in cancer cells.Oxygen availability strongly affects chronological lifespan and thermotolerance in batch cultures of Saccharomyces cerevisiae.Unconventional Targeting of a Thiol Peroxidase to the Mitochondrial Intermembrane Space Facilitates Oxidative Protein Folding.Amyloid-beta disrupts calcium and redox homeostasis in brain endothelial cells.FABP4/aP2 Regulates Macrophage Redox Signaling and Inflammasome Activation via Control of UCP2.Mitochondrial ROS versus ER ROS: Which Comes First in Myocardial Calcium Dysregulation?Cold-inducible RNA-binding protein through TLR4 signaling induces mitochondrial DNA fragmentation and regulates macrophage cell death after trauma.The role of flavin-containing enzymes in mitochondrial membrane hyperpolarization and ROS production in respiring Saccharomyces cerevisiae cells under heat-shock conditionsMicroRNA-21 regulates non-small cell lung cancer cell proliferation by affecting cell apoptosis via COX-19RasGRF Couples Nox4-Dependent Endoplasmic Reticulum Signaling to Ras.Effects of Disruption of PMC1 in the tfp1∆/∆ Mutant on Calcium Homeostasis, Oxidative and Osmotic Stress Resistance in Candida albicans.Partial involvement of Nrf2 in skeletal muscle mitohormesis as an adaptive response to mitochondrial uncoupling.Redox crosstalk at endoplasmic reticulum (ER) membrane contact sites (MCS) uses toxic waste to deliver messages.An Essential Role for ECSIT in Mitochondrial Complex I Assembly and Mitophagy in Macrophages.Mitochondrial dysfunction reduces yeast replicative lifespan by elevating RAS-dependent ROS production by the ER-localized NADPH oxidase Yno1.ER–Mitochondria Microdomains in Cardiac Ischemia–Reperfusion Injury: A Fresh Perspective.Lipotoxicty in yeast: a focus on plasma membrane signalling and membrane contact sites.
P2860
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P2860
Loss of cytochrome c oxidase promotes RAS-dependent ROS production from the ER resident NADPH oxidase, Yno1p, in yeast.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
@zh-hant
name
Loss of cytochrome c oxidase p ...... ADPH oxidase, Yno1p, in yeast.
@en
Loss of cytochrome c oxidase p ...... ADPH oxidase, Yno1p, in yeast.
@nl
type
label
Loss of cytochrome c oxidase p ...... ADPH oxidase, Yno1p, in yeast.
@en
Loss of cytochrome c oxidase p ...... ADPH oxidase, Yno1p, in yeast.
@nl
prefLabel
Loss of cytochrome c oxidase p ...... ADPH oxidase, Yno1p, in yeast.
@en
Loss of cytochrome c oxidase p ...... ADPH oxidase, Yno1p, in yeast.
@nl
P2093
P1433
P1476
Loss of cytochrome c oxidase p ...... ADPH oxidase, Yno1p, in yeast.
@en
P2093
Emma L Bastow
Geraldine Sanders
Jane E Leadsham
Michael Breitenbach
Rachael Hutton
Samantha Giannaki
Wesley R Naeimi
P304
P356
10.1016/J.CMET.2013.07.005
P577
2013-08-01T00:00:00Z