Effects of subchronic exposures to concentrated ambient particles (CAPs) in mice. II. The design of a CAPs exposure system for biometric telemetry monitoring.
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PM source apportionment for short-term cardiac function changes in ApoE-/- mice.Particulate matter-induced lung inflammation increases systemic levels of PAI-1 and activates coagulation through distinct mechanismsAirborne particulate matter selectively activates endoplasmic reticulum stress response in the lung and liver tissuesComparative effects of inhaled diesel exhaust and ambient fine particles on inflammation, atherosclerosis, and vascular dysfunctionInhalation toxicology methods: the generation and characterization of exposure atmospheres and inhalational exposuresInhaled nickel nanoparticles alter vascular reactivity in C57BL/6 miceAir pollution exposure potentiates hypertension through reactive oxygen species-mediated activation of Rho/ROCKAir quality in New York City hookah barsSource Apportionment and Elemental Composition of PM2.5 and PM10 in Jeddah City, Saudi ArabiaCardiovascular effects of nickel in ambient airβ₂-Adrenergic agonists augment air pollution-induced IL-6 release and thrombosisFine ambient air particulate matter exposure induces molecular alterations associated with vascular disease progression within plaques of atherosclerotic susceptible mice.Kupffer cell activation by ambient air particulate matter exposure may exacerbate non-alcoholic fatty liver disease.Particulate matter neurotoxicity in culture is size-dependentPulmonary response after exposure to inhaled nickel hydroxide nanoparticles: short and long-term studies in mice.Oxidant generation capacity of source-apportioned PM2.5.Real-world exposure of airborne particulate matter triggers oxidative stress in an animal modelAtherosclerosis lesion progression during inhalation exposure to environmental tobacco smoke: a comparison to concentrated ambient air fine particles exposure.Chronic fine particulate matter exposure induces systemic vascular dysfunction via NADPH oxidase and TLR4 pathwaysTranslocation and potential neurological effects of fine and ultrafine particles a critical update.Particulate matter Air Pollution induces hypermethylation of the p16 promoter Via a mitochondrial ROS-JNK-DNMT1 pathwayExposure to ambient particulate matter induces a NASH-like phenotype and impairs hepatic glucose metabolism in an animal model.Effect of co-exposure to nickel and particulate matter on insulin resistance and mitochondrial dysfunction in a mouse model.Exposure to fine airborne particulate matter induces macrophage infiltration, unfolded protein response, and lipid deposition in white adipose tissueCardiovascular remodeling in response to long-term exposure to fine particulate matter air pollutionComparative proteomics of inhaled silver nanoparticles in healthy and allergen provoked mice.Air pollution and cardiac remodeling: a role for RhoA/Rho-kinase.Exposure to fine airborne particulate matters induces hepatic fibrosis in murine models.Ambient air pollution exaggerates adipose inflammation and insulin resistance in a mouse model of diet-induced obesity.Long-term particulate matter exposure: Attributing health effects to individual PM components.Temporal variation of fine and coarse particulate matter sources in Jeddah, Saudi Arabia.Atherosclerosis and vasomotor dysfunction in arteries of animals after exposure to combustion-derived particulate matter or nanomaterials.Exposure to Concentrated Ambient PM2.5 Shortens Lifespan and Induces Inflammation-Associated Signaling and Oxidative Stress in Drosophila.The cellular and genomic response of an immortalized microglia cell line (BV2) to concentrated ambient particulate matter.Exposure to Ambient Particulate Matter during Specific Gestational Periods Produces Adverse Obstetric Consequences in Mice.Inhalation Exposure to PM2.5 Counteracts Hepatic Steatosis in Mice Fed High-fat Diet by Stimulating Hepatic Autophagy.Neuropathological Consequences of Gestational Exposure to Concentrated Ambient Fine and Ultrafine Particles in the Mouse.Perinatal exposure to concentrated ambient particulates results in autism-like behavioral deficits in adult mice.Exposure to fine and ultrafine particulate matter during gestation alters postnatal oligodendrocyte maturation, proliferation capacity, and myelination.Effects of subchronic exposures to concentrated ambient particles in mice. IX. Integral assessment and human health implications of subchronic exposures of mice to CAPs.
P2860
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P2860
Effects of subchronic exposures to concentrated ambient particles (CAPs) in mice. II. The design of a CAPs exposure system for biometric telemetry monitoring.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Effects of subchronic exposure ...... iometric telemetry monitoring.
@en
Effects of subchronic exposures to concentrated ambient particles
@nl
type
label
Effects of subchronic exposure ...... iometric telemetry monitoring.
@en
Effects of subchronic exposures to concentrated ambient particles
@nl
prefLabel
Effects of subchronic exposure ...... iometric telemetry monitoring.
@en
Effects of subchronic exposures to concentrated ambient particles
@nl
P2093
P2860
P1476
Effects of subchronic exposure ...... iometric telemetry monitoring.
@en
P2093
Christine Nadziejko
Judy Xiong
Mianhua Zhong
Polina Maciejczyk
P2860
P304
P356
10.1080/08958370590912743
P577
2005-04-01T00:00:00Z