Spinal TNF is necessary for inactivity-induced phrenic motor facilitation.
about
Decreased spinal synaptic inputs to phrenic motor neurons elicit localized inactivity-induced phrenic motor facilitation.Spinal NMDA receptor activation constrains inactivity-induced phrenic motor facilitation in Charles River Sprague-Dawley rats.Intermittent reductions in respiratory neural activity elicit spinal TNF-α-independent, atypical PKC-dependent inactivity-induced phrenic motor facilitation.Inactivity-induced respiratory plasticity: protecting the drive to breathe in disorders that reduce respiratory neural activityPlasticity in respiratory motor neurons in response to reduced synaptic inputs: A form of homeostatic plasticity in respiratory control?Reduced respiratory neural activity elicits a long-lasting decrease in the CO2 threshold for apnea in anesthetized rats.Intermittent apnea elicits inactivity-induced phrenic motor facilitation via a retinoic acid- and protein synthesis-dependent pathway.
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Spinal TNF is necessary for inactivity-induced phrenic motor facilitation.
description
2013 nî lūn-bûn
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Spinal TNF is necessary for inactivity-induced phrenic motor facilitation.
@en
Spinal TNF is necessary for inactivity-induced phrenic motor facilitation.
@nl
type
label
Spinal TNF is necessary for inactivity-induced phrenic motor facilitation.
@en
Spinal TNF is necessary for inactivity-induced phrenic motor facilitation.
@nl
prefLabel
Spinal TNF is necessary for inactivity-induced phrenic motor facilitation.
@en
Spinal TNF is necessary for inactivity-induced phrenic motor facilitation.
@nl
P2093
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P1476
Spinal TNF is necessary for inactivity-induced phrenic motor facilitation.
@en
P2093
Nathan A Baertsch
Oleg Broytman
Tracy L Baker-Herman
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P304
P356
10.1113/JPHYSIOL.2013.256644
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P577
2013-07-22T00:00:00Z