Diazoxide postconditioning induces mitochondrial protein S-nitrosylation and a redox-sensitive mitochondrial phosphorylation/translocation of RISK elements: no role for SAFE.
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The mechanism of Intralipid®-mediated cardioprotection complex IV inhibition by the active metabolite, palmitoylcarnitine, generates reactive oxygen species and activates reperfusion injury salvage kinasesThe role of gasotransmitters NO, H2S and CO in myocardial ischaemia/reperfusion injury and cardioprotection by preconditioning, postconditioning and remote conditioningIntermittent losartan administration triggers cardiac post-conditioning in isolated rat hearts: role of BK2 receptorsAdenosine A1 receptor activation increases myocardial protein S-nitrosothiols and elicits protection from ischemia-reperfusion injury in male and female heartsATP-sensitive potassium (KATP) channel openers diazoxide and nicorandil lower intraocular pressure by activating the Erk1/2 signaling pathwayToward a new STATe: the role of STATs in mitochondrial function.bFGF attenuates endoplasmic reticulum stress and mitochondrial injury on myocardial ischaemia/reperfusion via activation of PI3K/Akt/ERK1/2 pathway.Redox signalling and cardioprotection: translatability and mechanismExtracellular signalling molecules in the ischaemic/reperfused heart - druggable and translatable for cardioprotection?Additive cardioprotection by pharmacological postconditioning with hydrogen sulfide and nitric oxide donors in mouse heart: S-sulfhydration vs. S-nitrosylation.S-nitrosylation of TRIM72 mends the broken heart: a molecular modifier-mediated cardioprotection.Diazoxide Attenuates Postresuscitation Brain Injury in a Rat Model of Asphyxial Cardiac Arrest by Opening Mitochondrial ATP-Sensitive Potassium Channels.Postconditioning leads to an increase in protein S-nitrosylation.2,3,5,4'-Tetrahydroxystilbene-2-O-β-D-glucoside protects murine hearts against ischemia/reperfusion injury by activating Notch1/Hes1 signaling and attenuating endoplasmic reticulum stress.Redox balance and cardioprotection.Protein S-nitrosylation in preconditioning and postconditioning.Cardioprotection acquired through exercise: the role of ischemic preconditioning.Low abundance of mitochondrial DNA changes mitochondrial status and renders cells resistant to serum starvation and sodium nitroprusside insult.Ischemic postconditioning: mechanisms, comorbidities, and clinical application.The SAFE pathway for cardioprotection: is this a promising target?Cardioprotective kinase signaling to subsarcolemmal and interfibrillar mitochondria is mediated by caveolar structures.Ischemic postconditioning confers cardioprotection and prevents reduction of Trx-1 in young mice, but not in middle-aged and old mice.Genetic deletion of the adaptor protein p66Shc increases susceptibility to short-term ischaemic myocardial injury via intracellular salvage pathways.Mitochondria in Cardiac Postconditioning.
P2860
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P2860
Diazoxide postconditioning induces mitochondrial protein S-nitrosylation and a redox-sensitive mitochondrial phosphorylation/translocation of RISK elements: no role for SAFE.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
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name
Diazoxide postconditioning ind ...... SK elements: no role for SAFE.
@en
Diazoxide postconditioning ind ...... SK elements: no role for SAFE.
@nl
type
label
Diazoxide postconditioning ind ...... SK elements: no role for SAFE.
@en
Diazoxide postconditioning ind ...... SK elements: no role for SAFE.
@nl
prefLabel
Diazoxide postconditioning ind ...... SK elements: no role for SAFE.
@en
Diazoxide postconditioning ind ...... SK elements: no role for SAFE.
@nl
P2093
P2860
P1476
Diazoxide postconditioning ind ...... ISK elements: no role for SAFE
@en
P2093
P2860
P2888
P356
10.1007/S00395-013-0371-Z
P577
2013-07-20T00:00:00Z