Recombinant soluble apyrase APT102 inhibits thrombosis and intimal hyperplasia in vein grafts without adversely affecting hemostasis or re-endothelialization.

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Recombinant soluble apyrase APT102 inhibits thrombosis and intimal hyperplasia in vein grafts without adversely affecting hemostasis or re-endothelialization.

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http://www.wikidata.org/entity/Q47994855

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

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2017年學術文章

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2017年學術文章

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name

Recombinant soluble apyrase AP ...... asis or re-endothelialization.

@en

Recombinant soluble apyrase AP ...... asis or re-endothelialization.

@nl

type

Item

label

Recombinant soluble apyrase AP ...... asis or re-endothelialization.

@en

Recombinant soluble apyrase AP ...... asis or re-endothelialization.

@nl

prefLabel

Recombinant soluble apyrase AP ...... asis or re-endothelialization.

@en

Recombinant soluble apyrase AP ...... asis or re-endothelialization.

@nl

P1476

Recombinant soluble apyrase AP ...... asis or re-endothelialization.

@en

P2093

O Adeola

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R Chen

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S S Jeong

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T L Strawn

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W P Fay

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Y Ji

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P2860

The endothelial cell ecto-ADPase responsible for inhibition of platelet function is CD39.

The A2B adenosine receptor protects against inflammation and excessive vascular adhesion.

Combination treatment of r-tPA and an optimized human apyrase reduces mortality rate and hemorrhagic transformation 6h after ischemic stroke in aged female rats.

Extracellular nucleotide and nucleoside signaling in vascular and blood disease

Mouse vein graft hemodynamic manipulations to enhance experimental utility

Metabolic control of excessive extracellular nucleotide accumulation by CD39/ecto-nucleotidase-1: implications for ischemic vascular diseases.

Contribution of endothelial injury and inflammation in early phase to vein graft failure: the causal factors impact on the development of intimal hyperplasia in murine models.

Mouse model of venous bypass graft arteriosclerosis.

Coronary vein graft disease: pathogenesis and prevention.

Proliferative capacity of vein graft smooth muscle cells and fibroblasts in vitro correlates with graft stenosis.

P304

814-825

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P31

scholarly article

P356

10.1111/JTH.13621

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http://www.w3.org/2001/XMLSchema#string

P478

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P577

2017-02-23T00:00:00Z

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P698

28079982

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P921

thrombosis

P932

5378664

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