Deleting the HCN1 Subunit of Hyperpolarization-Activated Ion Channels in Mice Impairs Acoustic Startle Reflexes, Gap Detection, and Spatial Localization.

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Acoustic startle modification as a tool for evaluating auditory function of the mouse: Progress, pitfalls, and potential.Reflex Modification Audiometry Reveals Dual Roles for Olivocochlear Neurotransmission.Cellular Computations Underlying Detection of Gaps in Sounds and Lateralizing Sound Sources.

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P2860

Deleting the HCN1 Subunit of Hyperpolarization-Activated Ion Channels in Mice Impairs Acoustic Startle Reflexes, Gap Detection, and Spatial Localization.

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Deleting the HCN1 Subunit of H ...... ion, and Spatial Localization.

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Deleting the HCN1 Subunit of H ...... ion, and Spatial Localization.

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Deleting the HCN1 Subunit of H ...... ion, and Spatial Localization.

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Deleting the HCN1 Subunit of H ...... ion, and Spatial Localization.

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Deleting the HCN1 Subunit of H ...... ion, and Spatial Localization.

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Deleting the HCN1 Subunit of H ...... ion, and Spatial Localization.

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P1476

Deleting the HCN1 Subunit of H ...... ion, and Spatial Localization.

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P2093

James R Ison

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Paul D Allen

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P2860

Two heteromeric Kv1 potassium channels differentially regulate action potential firing

In the ventral cochlear nucleus Kv1.1 and subunits of HCN1 are colocalized at surfaces of neurons that have low-voltage-activated and hyperpolarization-activated conductances.

HCN1 and HCN2 proteins are expressed in cochlear hair cells: HCN1 can form a ternary complex with protocadherin 15 CD3 and F-actin-binding filamin A or can interact with HCN2

The superior paraolivary nucleus shapes temporal response properties of neurons in the inferior colliculus

Auditory deficits of Kcna1 deletion are similar to those of a monaural hearing impairment.

Structural Changes and Lack of HCN1 Channels in the Binaural Auditory Brainstem of the Naked Mole-Rat (Heterocephalus glaber).

The physiology of mechanoelectrical transduction channels in hearing.

Functional contributions of HCN channels in the primary auditory neurons of the mouse inner ear.

HCN channels expressed in the inner ear are necessary for normal balance function.

Origin and function of short-latency inputs to the neural substrates underlying the acoustic startle reflex

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s10162-016-0610-8

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scholarly article

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10.1007/S10162-016-0610-8

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Donata Oertel

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Deleting the HCN1 Subunit of Hyperpolarization-Activated Ion Channels in Mice Impairs Acoustic Startle Reflexes, Gap Detection, and Spatial Localization.

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P2860

P2860

Deleting the HCN1 Subunit of Hyperpolarization-Activated Ion Channels in Mice Impairs Acoustic Startle Reflexes, Gap Detection, and Spatial Localization.

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