PAR-1 and PAR-2 Expression Is Enhanced in Inflamed Odontoblast Cells.

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PAR-1 and PAR-2 Expression Is Enhanced in Inflamed Odontoblast Cells.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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PAR-1 and PAR-2 Expression Is Enhanced in Inflamed Odontoblast Cells.

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PAR-1 and PAR-2 Expression Is Enhanced in Inflamed Odontoblast Cells.

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PAR-1 and PAR-2 Expression Is Enhanced in Inflamed Odontoblast Cells.

@en

PAR-1 and PAR-2 Expression Is Enhanced in Inflamed Odontoblast Cells.

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PAR-1 and PAR-2 Expression Is Enhanced in Inflamed Odontoblast Cells.

@en

PAR-1 and PAR-2 Expression Is Enhanced in Inflamed Odontoblast Cells.

@nl

P1476

PAR-1 and PAR-2 Expression Is Enhanced in Inflamed Odontoblast Cells.

@en

P2093

F D Nascimento

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G E Moura

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G M Viana

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M M P Alvarez

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P2860

Mechanism of heparin acceleration of tissue inhibitor of metalloproteases-1 (TIMP-1) degradation by the human neutrophil elastase

Decreasing NF-κB expression enhances odontoblastic differentiation and collagen expression in dental pulp stem cells exposed to inflammatory cytokines.

Cysteine cathepsins in human carious dentin.

Differentiation of embryonic mesenchymal cells to odontoblast-like cells by overexpression of dentin matrix protein 1.

Protease-activated receptors: contribution to physiology and disease.

Enhanced Dentinogenesis of Pulp Progenitors by Early Exposure to FGF2.

Matrix metalloproteases and PAR1 activation.

Peripheral mechanisms of odontogenic pain.

Autophagy regulates odontoblast differentiation by suppressing NF-κB activation in an inflammatory environment.

Palmitoylation of protease-activated receptor-1 regulates adaptor protein complex-2 and -3 interaction with tyrosine-based motifs and endocytic sorting.

P304

1518-1525

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scholarly article

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10.1177/0022034517719415

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P407

English

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P478

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P50

Carlos Alberto de Souza Costa

Leo Tjäderhane

Ivarne L.S. Tersariol

Mauricio F.M. Machado

Helena B Nader

P577

2017-07-01T00:00:00Z

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P698

28759300

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