Hypothermia treatment potentiates ERK1/2 activation after traumatic brain injury.
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Mild hypothermia as a treatment for central nervous system injuries: Positive or negative effectsPathophysiology of astroglial purinergic signallingPhosphatidylinositol 3-kinase/protein kinase Cdelta activation induces close homolog of adhesion molecule L1 (CHL1) expression in cultured astrocytes.The evidence for hypothermia as a neuroprotectant in traumatic brain injuryPost-traumatic seizure susceptibility is attenuated by hypothermia therapy.Effects of hypothermia combined with neural stem cell transplantation on recovery of neurological function in rats with spinal cord injury.Hypothermic treatment for acute spinal cord injury.Mild Hypothermia Attenuates Intercellular Adhesion Molecule-1 Induction via Activation of Extracellular Signal-Regulated Kinase-1/2 in a Focal Cerebral Ischemia Model.Efficacy of mild hypothermia for the treatment of patients with cardiac arrest.Posttraumatic hypothermia increases doublecortin expressing neurons in the dentate gyrus after traumatic brain injury in the ratMolecular and cellular pathways as a target of therapeutic hypothermia: pharmacological aspectPhosphodiesterase isoform-specific expression induced by traumatic brain injury.Decoding hippocampal signaling deficits after traumatic brain injuryThe selective mGluR5 agonist CHPG protects against traumatic brain injury in vitro and in vivo via ERK and Akt pathwayImplication for long-term hypothermia on degradation of interleukin-8 mRNA in endothelial cells stimulated with lipopolysaccharides.Phosphodiesterase inhibition rescues chronic cognitive deficits induced by traumatic brain injuryTherapeutic hypothermia and targeted temperature management in traumatic brain injury: Clinical challenges for successful translationEmergence of cognitive deficits after mild traumatic brain injury due to hyperthermia.Protection in animal models of brain and spinal cord injury with mild to moderate hypothermia.Use of hypothermia in the intensive care unit.Phosphodiesterase inhibitors as therapeutics for traumatic brain injury.The role of cold-inducible RNA binding protein in cell stress response.Combination therapies for traumatic brain injury: prospective considerations.Hypothermia attenuates apoptosis and protects contact between myelin basic protein-expressing oligodendroglial-lineage cells and neurons against hypoxia-ischemia.The Profile of MMP-9, MMP-9 mRNA Expression, -1562 C/T Polymorphism and Outcome in High-risk Traumatic Brain Injury: The Effect of Therapeutic Mild Hypothermia.Therapeutic hypothermia and targeted temperature management for traumatic brain injury: Experimental and clinical experience
P2860
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P2860
Hypothermia treatment potentiates ERK1/2 activation after traumatic brain injury.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
Hypothermia treatment potentiates ERK1/2 activation after traumatic brain injury.
@en
Hypothermia treatment potentiates ERK1/2 activation after traumatic brain injury.
@nl
type
label
Hypothermia treatment potentiates ERK1/2 activation after traumatic brain injury.
@en
Hypothermia treatment potentiates ERK1/2 activation after traumatic brain injury.
@nl
prefLabel
Hypothermia treatment potentiates ERK1/2 activation after traumatic brain injury.
@en
Hypothermia treatment potentiates ERK1/2 activation after traumatic brain injury.
@nl
P2093
P2860
P1476
Hypothermia treatment potentiates ERK1/2 activation after traumatic brain injury.
@en
P2093
Anthony A Oliva
Bing-Ren Hu
Helen M Bramlett
Ofelia F Alonso
Shaoyi Chen
W Dalton Dietrich
P2860
P304
P356
10.1111/J.1460-9568.2007.05720.X
P407
P577
2007-07-30T00:00:00Z