Cardio-Oncology: mechanisms of cardiovascular toxicity.

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Cardio-Oncology: mechanisms of cardiovascular toxicity.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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2018年學術文章

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Cardio-Oncology: mechanisms of cardiovascular toxicity.

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Cardio-Oncology: mechanisms of cardiovascular toxicity.

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Cardio-Oncology: mechanisms of cardiovascular toxicity.

@en

Cardio-Oncology: mechanisms of cardiovascular toxicity.

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Cardio-Oncology: mechanisms of cardiovascular toxicity.

@en

Cardio-Oncology: mechanisms of cardiovascular toxicity.

@nl

P1476

Cardio-Oncology: mechanisms of cardiovascular toxicity

@en

P2093

Timothy M Markman

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Juvenile exposure to anthracyclines impairs cardiac progenitor cell function and vascularization resulting in greater susceptibility to stress-induced myocardial injury in adult mice

Raf-1 promotes cell survival by antagonizing apoptosis signal-regulating kinase 1 through a MEK-ERK independent mechanism

Selectivity and therapeutic inhibition of kinases: to be or not to be?

Conditional mutation of the ErbB2 (HER2) receptor in cardiomyocytes leads to dilated cardiomyopathy

Trastuzumab--mechanism of action and use in clinical practice

Cancer therapy-induced cardiotoxicity: basic mechanisms and potential cardioprotective therapies.

Deaths: final data for 2010.

Management of immune checkpoint blockade dysimmune toxicities: a collaborative position paper.

Hypertension and rarefaction during treatment with telatinib, a small molecule angiogenesis inhibitor.

Anthracycline-induced cardiomyopathy: clinical relevance and response to pharmacologic therapy.

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113

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scholarly article

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10.12688/F1000RESEARCH.12598.1

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P478

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Maurie Markman

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2018-01-25T00:00:00Z

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29399333

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5785712

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