Free radical reaction and biological defense mechanism in the pathogenesis of prolonged vasospasm in experimental subarachnoid hemorrhage.
about
Inhibition of nitric oxide generation and lipid peroxidation attenuates hemolysate-induced injury to cerebrovascular endothelium.Unfractionated heparin: multitargeted therapy for delayed neurological deficits induced by subarachnoid hemorrhage.Dexanabinol prevents development of vasospasm in the rat femoral artery model.The time course of intracranial pathophysiological changes following experimental subarachnoid haemorrhage in the rat.Alterations in serotonin and neuropeptide Y content of cerebrovascular sympathetic nerves following experimental subarachnoid hemorrhage.
P2860
Free radical reaction and biological defense mechanism in the pathogenesis of prolonged vasospasm in experimental subarachnoid hemorrhage.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年学术文章
@wuu
1988年学术文章
@zh-cn
1988年学术文章
@zh-hans
1988年学术文章
@zh-my
1988年学术文章
@zh-sg
1988年學術文章
@yue
1988年學術文章
@zh
1988年學術文章
@zh-hant
name
Free radical reaction and biol ...... ental subarachnoid hemorrhage.
@en
Free radical reaction and biol ...... ental subarachnoid hemorrhage.
@nl
type
label
Free radical reaction and biol ...... ental subarachnoid hemorrhage.
@en
Free radical reaction and biol ...... ental subarachnoid hemorrhage.
@nl
prefLabel
Free radical reaction and biol ...... ental subarachnoid hemorrhage.
@en
Free radical reaction and biol ...... ental subarachnoid hemorrhage.
@nl
P2093
P356
P1476
Free radical reaction and biol ...... ental subarachnoid hemorrhage.
@en
P2093
P356
10.1038/JCBFM.1988.1
P577
1988-02-01T00:00:00Z