Neuron-specific inactivation of the hypoxia inducible factor 1 alpha increases brain injury in a mouse model of transient focal cerebral ischemia.
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Hypoxia-inducible factors and the response to hypoxic stressHistone Deacetylases Exert Class-Specific Roles in Conditioning the Brain and Heart Against Acute Ischemic InjuryFrom rapid to delayed and remote postconditioning: the evolving concept of ischemic postconditioning in brain ischemiaTolerance to ischemia - an increasingly complex biologyNeuroprotection for stroke: current status and future perspectivesMolecular mechanisms of ischemic preconditioning in the kidneyThe ischemic environment drives microglia and macrophage functionMolecular mechanisms of action and therapeutic uses of pharmacological inhibitors of HIF-prolyl 4-hydroxylases for treatment of ischemic diseasesAdaptive cellular stress pathways as therapeutic targets of dietary phytochemicals: focus on the nervous systemChick embryo partial ischemia model: a new approach to study ischemia ex vivoDapagliflozin, SGLT2 Inhibitor, Attenuates Renal Ischemia-Reperfusion InjuryHIF-1 alpha is an essential effector for purine nucleoside-mediated neuroprotection against hypoxia in PC12 cells and primary cerebellar granule neuronsThe role of angiogenic factors in fibroid pathogenesis: potential implications for future therapyCombined antiapoptotic and antioxidant approach to acute neuroprotection for stroke in hypertensive rats.Hypoxia-induced transcription of dopamine D3 and D4 receptors in human neuroblastoma and astrocytoma cells.HIF-1 alpha-deficient mice have increased brain injury after neonatal hypoxia-ischemia.Impaired expression of neuroprotective molecules in the HIF-1alpha pathway following traumatic brain injury in aged mice.Ischemia induces different levels of hypoxia inducible factor-1α protein expression in interneurons and pyramidal neurons.Impaired Ca(2+)-handling in HIF-1alpha(+/-) mice as a consequence of pressure overload.Epigenetics and the environment: in search of the "toleroasome" vital to execution of ischemic preconditioning.Relative Contribution of Prolyl Hydroxylase-Dependent and -Independent Degradation of HIF-1alpha by Proteasomal Pathways in Cerebral Ischemia.Desferroxamine infusion increases cerebral blood flow: a potential association with hypoxia-inducible factor-1.Endothelial progenitor cells transplantation attenuated blood-brain barrier damage after ischemia in diabetic mice via HIF-1α.Expression of hypoxia-inducible factor 1 alpha and oligodendrocyte lineage gene-1 in cultured brain slices after oxygen-glucose deprivation.Inhibition of prolyl hydroxylases by dimethyloxaloylglycine after stroke reduces ischemic brain injury and requires hypoxia inducible factor-1α.Mitochondria: the missing link between preconditioning and neuroprotection.Differential effects of HIF-1 inhibition by YC-1 on the overall outcome and blood-brain barrier damage in a rat model of ischemic strokeHoushiheisan compound prescription protects neurovascular units after cerebral ischemia.MicroRNA in ischemic stroke etiology and pathology.Transient ureteral obstruction prevents against kidney ischemia/reperfusion injury via hypoxia-inducible factor (HIF)-2α activationHypoxic preconditioning protection is eliminated in HIF-1α knockout mice subjected to neonatal hypoxia-ischemia.Roles of individual prolyl-4-hydroxylase isoforms in the first 24 hours following transient focal cerebral ischaemia: insights from genetically modified mice.APP knockout mice experience acute mortality as the result of ischemia.Pharmacologic preconditioning: translating the promiseHuman embryonic stem cell neural differentiation and enhanced cell survival promoted by hypoxic preconditioningProphylactic neuroprotection against stroke: low-dose, prolonged treatment with deferoxamine or deferasirox establishes prolonged neuroprotection independent of HIF-1 function.O2 regulates stem cells through Wnt/β-catenin signalling.Disruption of hypoxia-inducible transcription factor-prolyl hydroxylase domain-1 (PHD-1-/-) attenuates ex vivo myocardial ischemia/reperfusion injury through hypoxia-inducible factor-1α transcription factor and its target genes in mice.Hypoxia-inducible factor-1α regulates the expression of L-type voltage-dependent Ca(2+) channels in PC12 cells under hypoxia.Erythropoietin in stroke therapy: friend or foe
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P2860
Neuron-specific inactivation of the hypoxia inducible factor 1 alpha increases brain injury in a mouse model of transient focal cerebral ischemia.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Neuron-specific inactivation o ...... sient focal cerebral ischemia.
@en
Neuron-specific inactivation o ...... sient focal cerebral ischemia.
@nl
type
label
Neuron-specific inactivation o ...... sient focal cerebral ischemia.
@en
Neuron-specific inactivation o ...... sient focal cerebral ischemia.
@nl
prefLabel
Neuron-specific inactivation o ...... sient focal cerebral ischemia.
@en
Neuron-specific inactivation o ...... sient focal cerebral ischemia.
@nl
P2093
P1476
Neuron-specific inactivation o ...... sient focal cerebral ischemia.
@en
P2093
Ioannis Dragatsis
Juan C Chavez
Luis F Miranda
Oxana Baranova
Paola Pichiule
P304
P356
10.1523/JNEUROSCI.0449-07.2007
P407
P577
2007-06-01T00:00:00Z