Siglec-9 is upregulated in rheumatoid arthritis and suppresses collagen-induced arthritis through reciprocal regulation of Th17-/Treg-cell differentiation.

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Yi Shen Juan Bi Pill Ameliorates Bone Loss and Destruction Induced by Arthritis Through Modulating the Balance of Cytokines Released by Different Subpopulations of T Cells.

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Siglec-9 is upregulated in rheumatoid arthritis and suppresses collagen-induced arthritis through reciprocal regulation of Th17-/Treg-cell differentiation.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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Siglec-9 is upregulated in rhe ...... 17-/Treg-cell differentiation.

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Siglec-9 is upregulated in rhe ...... 17-/Treg-cell differentiation.

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Siglec-9 is upregulated in rhe ...... 17-/Treg-cell differentiation.

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Siglec-9 is upregulated in rhe ...... 17-/Treg-cell differentiation.

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Siglec-9 is upregulated in rhe ...... 17-/Treg-cell differentiation.

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Siglec-9 is upregulated in rhe ...... 17-/Treg-cell differentiation.

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Siglec-9 is upregulated in rhe ...... h17-/Treg-cell differentiation

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D Liu

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H Shen

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J Liu

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Q Chen

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R Tu

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X Wang

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Y Ning

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Y Xiong

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Cloning, characterization, and phylogenetic analysis of siglec-9, a new member of the CD33-related group of siglecs. Evidence for co-evolution with sialic acid synthesis pathways

The American Rheumatism Association 1987 revised criteria for the classification of rheumatoid arthritis

Immunoreceptor tyrosine-based inhibition motifs: a quest in the past and future

The membrane-proximal immunoreceptor tyrosine-based inhibitory motif is critical for the inhibitory signaling mediated by Siglecs-7 and -9, CD33-related Siglecs expressed on human monocytes and NK cells

Genetics and epigenetics of rheumatoid arthritis

Soluble Siglec-9 suppresses arthritis in a collagen-induced arthritis mouse model and inhibits M1 activation of RAW264.7 macrophages

Siglec-9, a novel sialic acid binding member of the immunoglobulin superfamily expressed broadly on human blood leukocytes.

Identification and molecular characterization of a novel member of the siglec family (SIGLEC9).

Modified disease activity scores that include twenty-eight-joint counts. Development and validation in a prospective longitudinal study of patients with rheumatoid arthritis.

Role of siglecs and related glycan-binding proteins in immune responses and immunoregulation

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scholarly article

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10.1111/SJI.12543

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Siglec-9 is upregulated in rheumatoid arthritis and suppresses collagen-induced arthritis through reciprocal regulation of Th17-/Treg-cell differentiation.

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P2860

P2860

Siglec-9 is upregulated in rheumatoid arthritis and suppresses collagen-induced arthritis through reciprocal regulation of Th17-/Treg-cell differentiation.

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