Liver Regeneration Is Impaired in Mice with Acute Exposure to a Very Low Carbohydrate Diet.

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Liver Regeneration Is Impaired in Mice with Acute Exposure to a Very Low Carbohydrate Diet.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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Liver Regeneration Is Impaired ...... a Very Low Carbohydrate Diet.

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Liver Regeneration Is Impaired ...... a Very Low Carbohydrate Diet.

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Liver Regeneration Is Impaired ...... a Very Low Carbohydrate Diet.

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Liver Regeneration Is Impaired ...... a Very Low Carbohydrate Diet.

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Liver Regeneration Is Impaired ...... a Very Low Carbohydrate Diet.

@en

Liver Regeneration Is Impaired ...... a Very Low Carbohydrate Diet.

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P1476

Liver Regeneration Is Impaired ...... a Very Low Carbohydrate Diet.

@en

P2093

Hao Chen

http://www.w3.org/2001/XMLSchema#string

Wu Sun

http://www.w3.org/2001/XMLSchema#string

Yandie Lin

http://www.w3.org/2001/XMLSchema#string

Yun Cai

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P2860

Elucidating the metabolic regulation of liver regeneration

Nutritional modulation of liver regeneration by carbohydrates, lipids, and amino acids: a review.

The mitogen-activated protein kinase kinase/extracellular signal-regulated kinase cascade activation is a key signalling pathway involved in the regulation of G(1) phase progression in proliferating hepatocytes.

Impaired liver regeneration in Ldlr-/- mice is associated with an altered hepatic profile of cytokines, growth factors, and lipids.

Initiation of liver growth by tumor necrosis factor: deficient liver regeneration in mice lacking type I tumor necrosis factor receptor

Altered hepatic triglyceride content after partial hepatectomy without impaired liver regeneration in multiple murine genetic models.

Methionine and choline regulate the metabolic phenotype of a ketogenic diet.

Regeneration of liver with marked fatty change following partial hepatectomy in rats.

Steatosis is not sufficient to cause an impaired regenerative response after partial hepatectomy in rats.

Preoperative fasting improves survival after 90% hepatectomy.

P2888

s10620-017-4519-9

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1256-1264

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scholarly article

P356

10.1007/S10620-017-4519-9

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Hao Li

P577

2017-03-06T00:00:00Z

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P698

28265828

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