MTA-induced Notch activation enhances the proliferation of human dental pulp cells by inhibiting autophagic flux.

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MTA-induced Notch activation enhances the proliferation of human dental pulp cells by inhibiting autophagic flux.

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2017 nî lūn-bûn

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MTA-induced Notch activation e ...... by inhibiting autophagic flux.

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MTA-induced Notch activation e ...... by inhibiting autophagic flux.

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MTA-induced Notch activation e ...... by inhibiting autophagic flux.

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MTA-induced Notch activation e ...... by inhibiting autophagic flux.

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MTA-induced Notch activation e ...... by inhibiting autophagic flux.

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MTA-induced Notch activation e ...... by inhibiting autophagic flux.

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P1476

MTA-induced Notch activation e ...... by inhibiting autophagic flux

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P2860

Recognition and ubiquitination of Notch by Itch, a hect-type E3 ubiquitin ligase

Reactivation of Delta-Notch signaling after injury: complementary expression patterns of ligand and receptor in dental pulp

p62/SQSTM1 forms protein aggregates degraded by autophagy and has a protective effect on huntingtin-induced cell death

Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts

Postnatal human dental pulp stem cells (DPSCs) in vitro and in vivo

Clinical applications of mineral trioxide aggregate.

Suppression of MAPK/JNK-MTORC1 signaling leads to premature loss of organelles and nuclei by autophagy during terminal differentiation of lens fiber cells

mTOR inhibition activates overall protein degradation by the ubiquitin proteasome system as well as by autophagy.

The Notch pathway regulates both the proliferation and differentiation of follicular cells in the panoistic ovary of Blattella germanica.

Sealing ability of a mineral trioxide aggregate for repair of lateral root perforations.

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e52-e62

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scholarly article

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10.1111/IEJ.12811

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P478

50 Suppl 2

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Yaou Zhang

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2017-08-20T00:00:00Z

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28667754

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