Repetitive mild traumatic brain injury augments tau pathology and glial activation in aged hTau mice.
about
Neurobehavioral, neuropathological and biochemical profiles in a novel mouse model of co-morbid post-traumatic stress disorder and mild traumatic brain injury.Mechanical Stress as the Common Denominator between Chronic Inflammation, Cancer, and Alzheimer's DiseaseChronic traumatic encephalopathy: where are we and where are we going?Repetitive concussions in adolescent athletes - translating clinical and experimental research into perspectives on rehabilitation strategiesAcute and chronic traumatic encephalopathies: pathogenesis and biomarkers.Chronic traumatic encephalopathy: a spectrum of neuropathological changes following repetitive brain trauma in athletes and military personnelMolecular mechanisms of cognitive dysfunction following traumatic brain injuryChanges in the neurochemistry of athletes with repetitive brain trauma: preliminary results using localized correlated spectroscopyChronic Traumatic Encephalopathy in Athletes Involved with High-impact SportsChronic Repetitive Mild Traumatic Brain Injury Results in Reduced Cerebral Blood Flow, Axonal Injury, Gliosis, and Increased T-Tau and Tau OligomersChronic traumatic encephalopathy: clinical-biomarker correlations and current concepts in pathogenesis.The spectrum of neurobehavioral sequelae after repetitive mild traumatic brain injury: a novel mouse model of chronic traumatic encephalopathy.Microglial Activation in Traumatic Brain Injury.The pathophysiology underlying repetitive mild traumatic brain injury in a novel mouse model of chronic traumatic encephalopathy.The pathophysiology of repetitive concussive traumatic brain injury in experimental models; new developments and open questionsHumanized Tau Mice with Regionalized Amyloid Exhibit Behavioral Deficits but No Pathological Interaction.Repeated Closed Head Injury in Mice Results in Sustained Motor and Memory Deficits and Chronic Cellular Changes.Ccr2 deletion dissociates cavity size and tau pathology after mild traumatic brain injuryDendritic Spine Loss and Chronic White Matter Inflammation in a Mouse Model of Highly Repetitive Head Trauma.Rapid accumulation of endogenous tau oligomers in a rat model of traumatic brain injury: possible link between traumatic brain injury and sporadic tauopathiesAmyloid-β Peptides and Tau Protein as Biomarkers in Cerebrospinal and Interstitial Fluid Following Traumatic Brain Injury: A Review of Experimental and Clinical Studies.Brain injury, neuroinflammation and Alzheimer's disease.Sports-related concussions - media, science and policy.Blood-brain barrier and traumatic brain injury.Models of mild traumatic brain injury: translation of physiological and anatomic injury.Traumatic Brain Injury, Chronic Traumatic Encephalopathy, and Alzheimer's Disease: Common Pathologies Potentiated by Altered Zinc Homeostasis.Traumatic brain injury in hTau model mice: Enhanced acute macrophage response and altered long-term recovery.Noncontact Rotational Head Injury Produces Transient Cognitive Deficits but Lasting Neuropathological Changes.Histological evidence of chronic traumatic encephalopathy in a large series of neurodegenerative diseases.Repetitive mild traumatic brain injury causes optic nerve and retinal damage in a mouse model.The Biological Basis of Chronic Traumatic Encephalopathy following Blast Injury: A Literature Review.Practical considerations for choosing a mouse model of Alzheimer's disease.Inflammatory changes in optic nerve after closed-head repeated traumatic brain injury: Preliminary study.Negative Impact of Female Sex on Outcomes from Repetitive Mild Traumatic Brain Injury in hTau Mice Is Age Dependent: A Chronic Effects of Neurotrauma Consortium Study.Lifelong behavioral and neuropathological consequences of repetitive mild traumatic brain injury.Chronic traumatic encephalopathy-integration of canonical traumatic brain injury secondary injury mechanisms with tau pathology.Ratio of β-amyloid protein (Aβ) and Tau predicts the postoperative cognitive dysfunction on patients undergoing total hip/knee replacement surgery.Ultrastructural Changes in the White and Gray Matter of Mice at Chronic Time Points After Repeated Concussive Head Injury.Repeated Mild Traumatic Brain Injury: Potential Mechanisms of Damage.The Inflammatory Continuum of Traumatic Brain Injury and Alzheimer's Disease.
P2860
Q21131048-D873AB28-2455-4916-B810-A89C56351C26Q26781105-0D98F329-CB08-4303-BCCA-45A5EC0BEAAFQ26825263-B2288343-D486-43B6-B2CB-AB61522856C1Q26866488-5D5A51B7-A099-44DA-AE47-DD49D294EB45Q26995259-FD2FF569-78B8-4F77-99AB-F7FA5A8E4C7FQ27001265-61926F69-AAE0-4FF1-A0BE-EAF0B7F1C2B2Q28385324-8DB4C69B-A751-4E1D-8BE0-DB04FBA3D5BEQ28389930-0AF01587-ECB3-4A1E-8D50-D19336ED9C6EQ28393205-1F388F2C-81C5-48AD-B223-F9768B195687Q28397264-2BE9B9E3-6EC5-4673-83DD-95DA152D54AAQ30409194-4B883F31-98A0-477D-BFF8-2CE3CDF60CDCQ30582745-A3A9055F-022A-4AE7-BE82-89EDAE2A5FC7Q33843373-758E8A39-84A8-4D66-932C-3E5BE2D70C84Q34880054-6BEAD78F-F157-49E5-A94A-74BF126F6C23Q35706415-C1F1D7C2-CB48-4C73-BD46-4597D633338EQ35986968-64DB8540-D136-4501-98AF-582F65EF63E2Q36079903-564AE71A-989C-48F7-B2FF-50F1BAFB6268Q36346809-DA4B62C8-51B7-417C-8DB9-ACB56EB83032Q36753758-A9650EE1-992C-4CF5-AA63-8CECB56D80F5Q36910217-1E099230-0110-4649-897B-C4DAC3A17815Q36955963-37A6A17A-00E6-4901-9ED0-BE14BB30D3A4Q37001841-58B4FB5D-DFD9-433F-91C0-E2B75454451AQ37676914-38DE6F8E-9065-4587-B109-63B9CD4817A5Q38170329-94E2BA2B-363B-41A9-9413-ADE7B1702124Q38252018-13EDD80F-B867-4F39-B984-AD566E19C626Q38380370-C4E6AC6B-1F26-4DAD-9443-222E7CE97155Q38599516-3D9A15E6-33F2-4E7A-999E-638FA85F004FQ40134768-2D2E0F7F-9C73-4665-A33E-5E65259E32D3Q41189547-7A2B0260-2B77-449C-AB1F-E64C3223E707Q44383217-9C9B7DDA-4A8C-4EEF-A43B-59CDF0C2D5DDQ45068024-FFC987E9-25B7-4D6D-9A92-13B28DFC40B5Q47135235-CA90A293-1076-46AF-80D3-0E4D146C7DCDQ47366484-C6FED215-F08A-4A04-A941-E7B061E37017Q47556586-AE979722-0A28-4FB5-9803-822E1F0A91F0Q47644244-1E75883C-D573-4B35-8949-4C2CCF8C4CF3Q47887763-1F84F93B-CC25-4B26-B8E9-A2DA8426B056Q48506785-B466C173-108C-48F3-8999-5A47A366DE3DQ50709836-1294B62B-290A-4705-8B75-B8840421589AQ52086737-3984AFC4-6F70-4726-84CC-53A2D7D3744BQ53403638-ECDF734F-B383-47B8-99AF-16B4C5130BD1
P2860
Repetitive mild traumatic brain injury augments tau pathology and glial activation in aged hTau mice.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
@zh-hant
name
Repetitive mild traumatic brai ...... activation in aged hTau mice.
@en
Repetitive mild traumatic brai ...... activation in aged hTau mice.
@nl
type
label
Repetitive mild traumatic brai ...... activation in aged hTau mice.
@en
Repetitive mild traumatic brai ...... activation in aged hTau mice.
@nl
prefLabel
Repetitive mild traumatic brai ...... activation in aged hTau mice.
@en
Repetitive mild traumatic brai ...... activation in aged hTau mice.
@nl
P2093
P2860
P1476
Repetitive mild traumatic brai ...... activation in aged hTau mice.
@en
P2093
Benoit Mouzon
Corbin Bachmeier
Fiona Crawford
Joseph-Olubunmi Ojo
M Banks Greenberg
P2860
P304
P356
10.1097/NEN.0B013E3182814CDF
P577
2013-02-01T00:00:00Z