Altered energy balance causes selective changes in melanocortin-4(MC4-R), but not melanocortin-3 (MC3-R), receptors in specific hypothalamic regions: further evidence that activation of MC4-R is a physiological inhibitor of feeding.
about
Endocrine-disrupting chemicals: prepubertal exposures and effects on sexual maturation and thyroid activity in the female rat. A focus on the EDSTAC recommendations.The role of NPY in metabolic homeostasis: implications for obesity therapy.Relationship of arousal to circadian anticipatory behavior: ventromedial hypothalamus: one node in a hunger-arousal network.The regulation of feeding: a cross talk between peripheral and central signalling.New aspects of melanocortin signaling: a role for PRCP in α-MSH degradationEnergetic responses are triggered by caudal brainstem melanocortin receptor stimulation and mediated by local sympathetic effector circuits.From feeding one to feeding many: hormone-induced changes in bodyweight homeostasis during pregnancy.Neuronal control of energy homeostasisAgouti-related peptide and MC3/4 receptor agonists both inhibit excitatory hypothalamic ventromedial nucleus neuronsThe central melanocortin system affects the hypothalamo-pituitary thyroid axis and may mediate the effect of leptin.Distinct effects of leptin and a melanocortin receptor agonist injected into medial hypothalamic nuclei on glucose uptake in peripheral tissues.Cytokine signaling modulates blood-brain barrier function.Unraveling the central proopiomelanocortin neural circuits.Glycemic state regulates melanocortin, but not nesfatin-1, responsiveness of glucose-sensing neurons in the nucleus of the solitary tract.Ventromedial hypothalamic melanocortin receptor activation: regulation of activity energy expenditure and skeletal muscle thermogenesis.Leptin infusion and obesity in mouse cause alterations in the hypothalamic melanocortin system.Inhibitory Effect of the Melanocortin Receptor Agonist Melanotan-II (MTII) on Feeding Depends on Dietary Fat Content and not Obesity in Rats on Free-Choice Diets.Defense of Elevated Body Weight Setpoint in Diet-Induced Obese Rats on Low Energy Diet Is Mediated by Loss of Melanocortin Sensitivity in the Paraventricular Hypothalamic Nucleus.Chronic application of MTII in a rat model of obesity results in sustained weight loss.VMN/LHA functional inhibition in tumor-bearing rats suggests hypothalamic involvement in cancer anorexia.Anatomical characterization of bombesin receptor subtype-3 mRNA expression in the rodent central nervous system.Activity of the hypothalamic melanocortin system decreases in middle-aged and increases in old rats.Melanocortins and agouti-related protein modulate the excitability of two arcuate nucleus neuron populations by alteration of resting potassium conductances.Leptin directly acts within the hypothalamus to stimulate gonadotropin-releasing hormone secretion in vivo in rats.
P2860
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P2860
Altered energy balance causes selective changes in melanocortin-4(MC4-R), but not melanocortin-3 (MC3-R), receptors in specific hypothalamic regions: further evidence that activation of MC4-R is a physiological inhibitor of feeding.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
Altered energy balance causes ...... ological inhibitor of feeding.
@en
Altered energy balance causes ...... ological inhibitor of feeding.
@nl
type
label
Altered energy balance causes ...... ological inhibitor of feeding.
@en
Altered energy balance causes ...... ological inhibitor of feeding.
@nl
prefLabel
Altered energy balance causes ...... ological inhibitor of feeding.
@en
Altered energy balance causes ...... ological inhibitor of feeding.
@nl
P2093
P1433
P1476
Altered energy balance causes ...... ological inhibitor of feeding.
@en
P2093
Harrold JA
Widdowson PS
Williams G
P304
P356
10.2337/DIABETES.48.2.267
P407
P577
1999-02-01T00:00:00Z