SEMA3C drives cancer growth by transactivating multiple receptor tyrosine kinases via Plexin B1.

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Semaphorin 3C and Its Receptors in Cancer and Cancer Stem-Like Cells.The genomic landscape of cutaneous SCC reveals drivers and a novel azathioprine associated mutational signature

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SEMA3C drives cancer growth by transactivating multiple receptor tyrosine kinases via Plexin B1.

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2018 nî lūn-bûn

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2018年の論文

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SEMA3C drives cancer growth by ...... yrosine kinases via Plexin B1.

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SEMA3C drives cancer growth by ...... yrosine kinases via Plexin B1.

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SEMA3C drives cancer growth by ...... yrosine kinases via Plexin B1.

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SEMA3C drives cancer growth by ...... yrosine kinases via Plexin B1.

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SEMA3C drives cancer growth by ...... yrosine kinases via Plexin B1.

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SEMA3C drives cancer growth by ...... yrosine kinases via Plexin B1.

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SEMA3C drives cancer growth by ...... tyrosine kinases via Plexin B1

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Alice L-F Mui

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Amina Zoubeidi

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Ario Takeuchi

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Daksh Thaper

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Daniel Hf Hui

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Hidetoshi Kuruma

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Ivy Zf Jiao

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James W Peacock

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Kevin Ck Lee

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Kevin J Tam

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Clusterin facilitates COMMD1 and I-kappaB degradation to enhance NF-kappaB activity in prostate cancer cells

EGF activates its receptor by removing interactions that autoinhibit ectodomain dimerization

Identification of semaphorin E as a non-MDR drug resistance gene of human cancers

MET amplification occurs with or without T790M mutations in EGFR mutant lung tumors with acquired resistance to gefitinib or erlotinib

NCBI GEO: mining tens of millions of expression profiles--database and tools update

Plexin-B1/RhoGEF-mediated RhoA activation involves the receptor tyrosine kinase ErbB-2

Growth factor and signaling pathways and their relevance to prostate cancer therapeutics

Targeting the adaptive molecular landscape of castration-resistant prostate cancer

Semaphorin signaling in cardiovascular development

Functional regulation of semaphorin receptors by proprotein convertases

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scholarly article

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10.15252/EMMM.201707689

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SEMA3C drives cancer growth by transactivating multiple receptor tyrosine kinases via Plexin B1.

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SEMA3C drives cancer growth by transactivating multiple receptor tyrosine kinases via Plexin B1.

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