Stimulatory effects of protein kinase C and calmodulin kinase II on N-methyl-D-aspartate receptor/channels in the postsynaptic density of rat brain.
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Ca2+/calmodulin-dependent protein kinase II alpha is required for the initiation and maintenance of opioid-induced hyperalgesiaKnockdown of spinal metabotropic glutamate receptor 1 (mGluR(1)) alleviates pain and restores opioid efficacy after nerve injury in ratsTransition from reversible to persistent binding of CaMKII to postsynaptic sites and NR2B.Haloperidol disrupts opioid-antinociceptive tolerance and physical dependenceCurcumin attenuates opioid tolerance and dependence by inhibiting Ca2+/calmodulin-dependent protein kinase II α activityNucleotides and phosphorylation bi-directionally modulate Ca2+/calmodulin-dependent protein kinase II (CaMKII) binding to the N-methyl-D-aspartate (NMDA) receptor subunit GluN2B.Calcium/calmodulin kinase II in the pedunculopontine tegmental nucleus modulates the initiation and maintenance of wakefulnessBrain-derived neurotrophic factor rapidly enhances phosphorylation of the postsynaptic N-methyl-D-aspartate receptor subunit 1Protein kinase C in pain: involvement of multiple isoformsInnate differences in protein expression in the nucleus accumbens and hippocampus of inbred alcohol-preferring and -nonpreferring rats.siRNA capsulated brain-targeted nanoparticles specifically knock down OATP2B1 in mice: a mechanism for acute morphine tolerance suppressionAcute inhibition of Ca2+/calmodulin-dependent protein kinase II reverses experimental neuropathic pain in mice.Normal and abnormal calcium homeostasis in neurons: a basis for the pathophysiology of traumatic and ischemic central nervous system injury.Neuronal plasticity in the cingulate cortex of rats following esophageal acid exposure in early life.Is persistent activity of calcium/calmodulin-dependent kinase required for the maintenance of LTP?CaMKIIα underlies spontaneous and evoked pain behaviors in Berkeley sickle cell transgenic mice.CaMKIIα Mediates the Effect of IL-17 To Promote Ongoing Spontaneous and Evoked Pain in Multiple Sclerosis.Hypothermia inhibits translocation of CaM kinase II and PKC-alpha, beta, gamma isoforms and fodrin proteolysis in rat brain synaptosome during ischemia-reperfusion.
P2860
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P2860
Stimulatory effects of protein kinase C and calmodulin kinase II on N-methyl-D-aspartate receptor/channels in the postsynaptic density of rat brain.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
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1993年學術文章
@zh-hant
name
Stimulatory effects of protein ...... synaptic density of rat brain.
@en
Stimulatory effects of protein ...... synaptic density of rat brain.
@nl
type
label
Stimulatory effects of protein ...... synaptic density of rat brain.
@en
Stimulatory effects of protein ...... synaptic density of rat brain.
@nl
prefLabel
Stimulatory effects of protein ...... synaptic density of rat brain.
@en
Stimulatory effects of protein ...... synaptic density of rat brain.
@nl
P2093
P2860
P1476
Stimulatory effects of protein ...... synaptic density of rat brain.
@en
P2093
P2860
P304
P356
10.1111/J.1471-4159.1993.TB03542.X
P407
P577
1993-07-01T00:00:00Z