Abnormal glutamate receptor expression in the medial temporal lobe in schizophrenia and mood disorders. - Wikidata - wikimedia - TriplyDB

Abnormal glutamate receptor expression in the medial temporal lobe in schizophrenia and mood disorders.

Abnormal glutamate receptor expression in the medial temporal lobe in schizophrenia and mood disorders.

http://www.wikidata.org/entity/Q48273349

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Bipolar disorder and mechanisms of action of mood stabilizers GABA neurons and the mechanisms of network oscillations: implications for understanding cortical dysfunction in schizophrenia In Sickness and in Health: Perineuronal Nets and Synaptic Plasticity in Psychiatric Disorders A Role for Estrogen in Schizophrenia: Clinical and Preclinical Findings A brief history of the development of antidepressant drugs: from monoamines to glutamate The Role of Hippocampal NMDA Receptors in Long-Term Emotional Responses following Muscarinic Receptor Activation Binding site and ligand flexibility revealed by high resolution crystal structures of GluK1 competitive antagonists In vivo assessment of neurotransmitters and modulators with magnetic resonance spectroscopy: application to schizophrenia The tyrosine phosphatase STEP: implications in schizophrenia and the molecular mechanism underlying antipsychotic medications Deletion of glutamate delta-1 receptor in mouse leads to aberrant emotional and social behaviors Metabotropic glutamate receptor 3 is associated with heroin dependence but not depression or schizophrenia in a Chinese population Regulation of kainate receptor subunit mRNA by stress and corticosteroids in the rat hippocampus Schizophrenia: redox regulation and volume neurotransmission Modeling mania in preclinical settings: A comprehensive review.Glutamatergic synaptic dysregulation in schizophrenia: therapeutic implications.Chronic ketamine produces altered distribution of parvalbumin-positive cells in the hippocampus of adult rats.GABA transporter-1 deficiency confers schizophrenia-like behavioral phenotypes.Reduced expression of the NMDA receptor-interacting protein SynGAP causes behavioral abnormalities that model symptoms of Schizophrenia Is lead exposure in early life an environmental risk factor for Schizophrenia? Neurobiological connections and testable hypotheses.Effects of the selective kainate receptor antagonist ACET on altered sensorimotor gating in a genetic model of reduced NMDA receptor function Nicotine ameliorates NMDA receptor antagonist-induced deficits in contextual fear conditioning through high-affinity nicotinic acetylcholine receptors in the hippocampus.Auditory steady state response in bipolar disorder: relation to clinical state, cognitive performance, medication status, and substance disorders Fluoxetine affects GluK2 editing, glutamate-evoked Ca(2+) influx and extracellular signal-regulated kinase phosphorylation in mouse astrocytes.Increased sensitivity to kainic acid in a genetic model of reduced NMDA receptor function.Reverse translational strategies for developing animal models of bipolar disorder.Remodeling of axo-spinous synapses in the pathophysiology and treatment of depression.Bimodal effect of lithium plasma levels on hippocampal glutamate concentrations in bipolar II depression: a pilot study.Statistical evaluation of methods for quantifying gene expression by autoradiography in histological sections.Evidence for the involvement of the kainate receptor subunit GluR6 (GRIK2) in mediating behavioral displays related to behavioral symptoms of mania Mood-stabilizers target the brain arachidonic acid cascade.N-methyl-D-aspartate receptor expression in parvalbumin-containing inhibitory neurons in the prefrontal cortex in bipolar disorder Antidepressant effects of ketamine and the roles of AMPA glutamate receptors and other mechanisms beyond NMDA receptor antagonism Expression of the NR2B-NMDA receptor trafficking complex in prefrontal cortex from a group of elderly patients with schizophrenia.Glutamatergic gene expression is specifically reduced in thalamocortical projecting relay neurons in schizophrenia.Proton magnetic resonance spectroscopy in 22q11 deletion syndrome.Polymorphisms in the GRIA1 gene region in psychotic bipolar disorder.High novelty-seeking rats are resilient to negative physiological effects of the early life stress.The role of rodent models in the discovery of new treatments for schizophrenia: updating our strategy.Does gene deletion of AMPA GluA1 phenocopy features of schizoaffective disorder?GluN2B protein deficits in the left, but not the right, hippocampus in schizophrenia.

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Abnormal glutamate receptor expression in the medial temporal lobe in schizophrenia and mood disorders.

http://www.wikidata.org/entity/Q48273349

description

2007 nî lūn-bûn

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Abnormal glutamate receptor ex ...... izophrenia and mood disorders.

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Abnormal glutamate receptor ex ...... izophrenia and mood disorders.

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Abnormal glutamate receptor ex ...... izophrenia and mood disorders.

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Abnormal glutamate receptor ex ...... izophrenia and mood disorders.

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Abnormal glutamate receptor ex ...... izophrenia and mood disorders.

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Abnormal glutamate receptor ex ...... izophrenia and mood disorders.

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Neuropsychopharmacology

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Abnormal glutamate receptor ex ...... hizophrenia and mood disorders

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Akinwunmi Oni-Orisan

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James H Meador-Woodruff

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Monica Beneyto

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Robert E McCullumsmith

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P2860

Meta-analysis of regional brain volumes in schizophrenia.

Limbic system abnormalities identified in schizophrenia using positron emission tomography with fluorodeoxyglucose and neocortical alterations with deficit syndrome.

The medial temporal lobe in schizophrenia.

The thalamus as a site of action of antipsychotic drugs.

Sequence determinants on the NR2A and NR2B subunits of NMDA receptor responsible for specificity of phosphorylation by CaMKII.

Regionally specific pattern of neurochemical pathology in schizophrenia as assessed by multislice proton magnetic resonance spectroscopic imaging.

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1888-1902

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scholarly article

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10.1038/SJ.NPP.1301312

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9

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Lars V Kristiansen

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2007-02-14T00:00:00Z

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