Suppression of hypoglossal motoneurons during the carbachol-induced atonia of REM sleep is not caused by fast synaptic inhibition.
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The neuropharmacology of upper airway motor control in the awake and asleep states: implications for obstructive sleep apnoeaRevisiting Antagonist Effects in Hypoglossal Nucleus: Brainstem Circuit for the State-Dependent Control of Hypoglossal Motoneurons: A HypothesisMicrodialysis perfusion of 5-HT into hypoglossal motor nucleus differentially modulates genioglossus activity across natural sleep-wake states in ratsSynaptic control of motoneuronal excitability.Pathophysiology & genetics of obstructive sleep apnoeaMechanical properties of the upper airway.Cholinergic modulation of respiratory brain-stem neurons and its function in sleep-wake state determination.Postsynaptic inhibition of hypoglossal motoneurons produces atonia of the genioglossal muscle during rapid eye movement sleep.Evidence that adrenergic ventrolateral medullary cells are activated whereas precerebellar lateral reticular nucleus neurons are suppressed during REM sleep.Sleep-wake control of the upper airway by noradrenergic neurons, with and without intermittent hypoxia.Serotonergic mechanisms in amyotrophic lateral sclerosis.Respiratory motor activity: influence of neuromodulators and implications for sleep disordered breathing.Adventures and tribulations in the search for the mechanisms of the atonia of REM sleep.Electromyographic activity at the base and tip of the tongue across sleep-wake states in rats.Quantitative differences among EMG activities of muscles innervated by subpopulations of hypoglossal and upper spinal motoneurons during non-REM sleep - REM sleep transitions: a window on neural processes in the sleeping brain.Neural Control of the Upper Airway: Respiratory and State-Dependent Mechanisms.Catecholaminergic A1/C1 neurons contribute to the maintenance of upper airway muscle tone but may not participate in NREM sleep-related depression of these muscles.REM sleep-like atonia of hypoglossal (XII) motoneurons is caused by loss of noradrenergic and serotonergic inputs.Unraveling the mechanisms of REM sleep atonia.Prenatal nicotine exposure increases apnoea and reduces nicotinic potentiation of hypoglossal inspiratory output in mice.Role of inhibitory amino acids in control of hypoglossal motor outflow to genioglossus muscle in naturally sleeping rats.GABAA receptor antagonism at the hypoglossal motor nucleus increases genioglossus muscle activity in NREM but not REM sleep.Are all motoneurons created equal in the eyes of REM sleep and the mechanisms of muscle atonia?Glycine-mediated postsynaptic inhibition is responsible for REM sleep atonia.Glycine at hypoglossal motor nucleus: genioglossus activity, CO(2) responses, and the additive effects of GABA.The modulation by 5-HT of glutamatergic inputs from the raphe pallidus to rat hypoglossal motoneurones, in vitro.Electrophysiological properties of lumbar motoneurons in the alpha-chloralose-anesthetized cat during carbachol-induced motor inhibition.Noradrenergic, serotonergic and GABAergic antagonists injected together into the XII nucleus abolish the REM sleep-like depression of hypoglossal motoneuronal activity.Duloxetine-induced rapid eye movement sleep behavior disorder: a case report.The pathogenesis of obstructive sleep apnea: advances in the past 100 years.The anatomical, cellular and synaptic basis of motor atonia during rapid eye movement sleep.Control of hypoglossal motoneurones during naturally occurring sleep and wakefulness in the intact, unanaesthetized cat: a field potential study.Identification of the mechanism mediating genioglossus muscle suppression in REM sleep.Development of glycinergic synaptic transmission to rat brain stem motoneurons.Facial muscle activation during sleep and its relation to the rapid eye movements of REM sleep.Effects of sleep-wake state on the genioglossus vs.diaphragm muscle response to CO(2) in rats.Control of Upper Airway Motoneurons During REM Sleep.Endogenous excitatory drive to the respiratory system in rapid eye movement sleep in cats.Computational model of brainstem circuit for state-dependent control of hypoglossal motoneurons.Neuroanatomical Basis of State-Dependent Activity of Upper Airway Muscles
P2860
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P2860
Suppression of hypoglossal motoneurons during the carbachol-induced atonia of REM sleep is not caused by fast synaptic inhibition.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh-hant
name
Suppression of hypoglossal mot ...... d by fast synaptic inhibition.
@en
Suppression of hypoglossal mot ...... d by fast synaptic inhibition.
@nl
type
label
Suppression of hypoglossal mot ...... d by fast synaptic inhibition.
@en
Suppression of hypoglossal mot ...... d by fast synaptic inhibition.
@nl
prefLabel
Suppression of hypoglossal mot ...... d by fast synaptic inhibition.
@en
Suppression of hypoglossal mot ...... d by fast synaptic inhibition.
@nl
P2093
P1433
P1476
Suppression of hypoglossal mot ...... d by fast synaptic inhibition.
@en
P2093
P304
P356
10.1016/0006-8993(93)90517-Q
P407
P577
1993-05-01T00:00:00Z