The C3aR promotes macrophage infiltration and regulates ANCA production but does not affect glomerular injury in experimental anti-myeloperoxidase glomerulonephritis.

about

The C3aR promotes macrophage infiltration and regulates ANCA production but does not affect glomerular injury in experimental anti-myeloperoxidase glomerulonephritis.

Item

http://www.wikidata.org/entity/Q48284080

description

2018 nî lūn-bûn

@nan

2018年の論文

@ja

2018年学术文章

@wuu

2018年学术文章

@zh

2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

@yue

2018年學術文章

@zh-hant

name

The C3aR promotes macrophage i ...... peroxidase glomerulonephritis.

@en

The C3aR promotes macrophage i ...... peroxidase glomerulonephritis.

@nl

type

Item

label

The C3aR promotes macrophage i ...... peroxidase glomerulonephritis.

@en

The C3aR promotes macrophage i ...... peroxidase glomerulonephritis.

@nl

prefLabel

The C3aR promotes macrophage i ...... peroxidase glomerulonephritis.

@en

The C3aR promotes macrophage i ...... peroxidase glomerulonephritis.

@nl

P1476

The C3aR promotes macrophage i ...... operoxidase glomerulonephritis

@en

P2093

Poh-Yi Gan

http://www.w3.org/2001/XMLSchema#string

Stephen R Holdsworth

http://www.w3.org/2001/XMLSchema#string

P2860

Chimeric receptors of the human C3a receptor and C5a receptor (CD88)

Antineutrophil cytoplasmic autoantibodies specific for myeloperoxidase cause glomerulonephritis and vasculitis in mice

The human C3a receptor is expressed on neutrophils and monocytes, but not on B or T lymphocytes

Association of Serum C3 Concentration and Histologic Signs of Thrombotic Microangiopathy with Outcomes among Patients with ANCA-Associated Renal Vasculitis.

Th17 cells promote autoimmune anti-myeloperoxidase glomerulonephritis.

Absence of signaling into CD4⁺ cells via C3aR and C5aR enables autoinductive TGF-β1 signaling and induction of Foxp3⁺ regulatory T cells.

Role of C3a receptors, C5a receptors, and complement protein C6 deficiency in collagen antibody-induced arthritis in mice.

Complement modulation of T cell immune responses during homeostasis and disease.

Negative regulation of pulmonary Th17 responses by C3a anaphylatoxin during allergic inflammation in mice.

Aggravation of anti-myeloperoxidase antibody-induced glomerulonephritis by bacterial lipopolysaccharide: role of tumor necrosis factor-alpha.

P304

e0190655

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1371/JOURNAL.PONE.0190655

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

A Richard Kitching

Jonathan Dick

P577

2018-01-09T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29315316

http://www.w3.org/2001/XMLSchema#string

P932

5760037

http://www.w3.org/2001/XMLSchema#string