Cocaine-induced expression of striatal c-fos in the rat is inhibited by NMDA receptor antagonists.
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Addiction-related gene regulation: risks of exposure to cognitive enhancers vs. other psychostimulantsPsychomotor stimulant- and opiate-induced c-fos mRNA expression patterns in the rat forebrain: comparisons between acute drug treatment and a drug challenge in sensitized animalsEffects of lamotrigine on behavioral and cardiovascular responses to cocaine in human subjectsChronic methamphetamine exposure suppresses the striatal expression of members of multiple families of immediate early genes (IEGs) in the rat: normalization by an acute methamphetamine injectionInfluence of glucocorticoids on dopaminergic transmission in the rat dorsolateral striatum.The rate of intravenous cocaine administration alters c-fos mRNA expression and the temporal dynamics of dopamine, but not glutamate, overflow in the striatum.Regulation of neurotransmitter interactions in the ventral striatum.Selective serotonin reuptake inhibitor antidepressants potentiate methylphenidate (Ritalin)-induced gene regulation in the adolescent striatum.Dopamine D1 receptors mediate CREB phosphorylation via phosphorylation of the NMDA receptor at Ser897-NR1.Cocaine increases phosphorylation of MeCP2 in the rat striatum in vivo: a differential role of NMDA receptors.Using c-fos to study neuronal ensembles in corticostriatal circuitry of addiction.Region-specific effects of isoflurane anesthesia on Fos immunoreactivity in response to intravenous cocaine challenge in rats with a history of repeated cocaine administrationCocaine-induced Fos expression is detectable in the frontal cortex and striatum of rats under isoflurane but not alpha-chloralose anesthesia: implications for FMRI.Cocaethylene: effects on brain systems and behavior.Cocaine-induced changes in NMDA receptor signaling.Amphetamine and dopamine-induced immediate early gene expression in striatal neurons depends on postsynaptic NMDA receptors and calcium.NMDAR dependent intracellular responses associated with cocaine conditioned place preference behavior.Effects of dopamine and NMDA receptors on cocaine-induced Fos expression in the striatum of Fischer ratsTopography of cocaine-induced gene regulation in the rat striatum: relationship to cortical inputs and role of behavioural context.Relapse to cocaine-seeking increases activity-regulated gene expression differentially in the striatum and cerebral cortex of rats following short or long periods of abstinence.Genetic NMDA receptor deficiency disrupts acute and chronic effects of cocaine but not amphetamine.Interaction between the serotonergic, dopaminergic, and glutamatergic systems in fenfluramine-induced Fos expression in striatal neurons.The activity-regulated cytoskeletal-associated protein arc is expressed in different striosome-matrix patterns following exposure to amphetamine and cocaine.Differential behavioral responses to cocaethylene of Long-Evans and Sprague-Dawley rats: role of serotonin.
P2860
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P2860
Cocaine-induced expression of striatal c-fos in the rat is inhibited by NMDA receptor antagonists.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
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1993年学术文章
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1993年学术文章
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1993年学术文章
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1993年学术文章
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1993年學術文章
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name
Cocaine-induced expression of ...... by NMDA receptor antagonists.
@en
Cocaine-induced expression of ...... by NMDA receptor antagonists.
@nl
type
label
Cocaine-induced expression of ...... by NMDA receptor antagonists.
@en
Cocaine-induced expression of ...... by NMDA receptor antagonists.
@nl
prefLabel
Cocaine-induced expression of ...... by NMDA receptor antagonists.
@en
Cocaine-induced expression of ...... by NMDA receptor antagonists.
@nl
P1476
Cocaine-induced expression of ...... by NMDA receptor antagonists.
@en
P2093
P304
P356
10.1016/0361-9230(93)90055-G
P577
1993-01-01T00:00:00Z