Role of complement C5 and T lymphocytes in pathogenesis of disseminated and mucosal candidiasis in susceptible DBA/2 mice. - Wikidata - wikimedia - TriplyDB

Role of complement C5 and T lymphocytes in pathogenesis of disseminated and mucosal candidiasis in susceptible DBA/2 mice.

Role of complement C5 and T lymphocytes in pathogenesis of disseminated and mucosal candidiasis in susceptible DBA/2 mice.

http://www.wikidata.org/entity/Q48371414

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Mucosal damage and neutropenia are required for Candida albicans dissemination Novel insights into disseminated candidiasis: pathogenesis research and clinical experience converge Genetic control of susceptibility to infection with Candida albicans in mice.IL-12 and related cytokines: function and regulatory implications in Candida albicans infection Genetic analysis of innate immunity in resistance to Candida albicans.Active immunizations with peptide-DC vaccines and passive transfer with antibodies protect neutropenic mice against disseminated candidiasis Use of a genetically engineered strain to evaluate the pathogenic potential of yeast cell and filamentous forms during Candida albicans systemic infection in immunodeficient mice.Animal models: an important tool in mycology.Efficacy of a genetically engineered Candida albicans tet-NRG1 strain as an experimental live attenuated vaccine against hematogenously disseminated candidiasis.Dysregulated inflammatory response to Candida albicans in a C5-deficient mouse strain.Th17 cells in immunity to Candida albicans.The aryl hydrocarbon receptor in innate T cell immunity.Therapeutic Effects of Monoclonal Antibody against Dengue Virus NS1 in a STAT1 Knockout Mouse Model of Dengue Infection.Next-generation computational genetic analysis: multiple complement alleles control survival after Candida albicans infection.Temporal events in the intravenous challenge model for experimental Candida albicans infections in female mice.Resistance and Susceptibility to Malarial Infection: A Host Defense Strategy against Malaria Genetic control of susceptibility to Candida albicans in susceptible A/J and resistant C57BL/6J mice.Reasons why DBA/2 mice are resistant to malarial infection: expansion of CD3int B220+ gammadelta T cells with double-negative CD4- CD8- phenotype in the liver.Fungal Strategies to Evade the Host Immune Recognition.Innate versus adaptive immunity in Candida albicans infection.Candida Immunity

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Role of complement C5 and T lymphocytes in pathogenesis of disseminated and mucosal candidiasis in susceptible DBA/2 mice.

http://www.wikidata.org/entity/Q48371414

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

@zh-my

2003年学术文章

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2003年學術文章

@yue

2003年學術文章

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name

Role of complement C5 and T ly ...... sis in susceptible DBA/2 mice.

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Role of complement C5 and T ly ...... sis in susceptible DBA/2 mice.

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type

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Role of complement C5 and T ly ...... sis in susceptible DBA/2 mice.

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Role of complement C5 and T ly ...... sis in susceptible DBA/2 mice.

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Role of complement C5 and T ly ...... sis in susceptible DBA/2 mice.

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Role of complement C5 and T ly ...... sis in susceptible DBA/2 mice.

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Microbial Pathogenesis

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Role of complement C5 and T ly ...... sis in susceptible DBA/2 mice.

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Alma Fulurija

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Camile S Farah

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John M Papadimitriou

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Karen E Drysdale

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Owen Naidoo

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Robert B Ashman

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Theo Gotjamanos

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103-113

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2

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