High-energy compounds promote physiological processing of Alzheimer's amyloid-β precursor protein and boost cell survival in culture.
about
Our "energy-Ca(2+) signaling deficits" hypothesis and its explanatory potential for key features of Alzheimer's disease.CNF1 increases brain energy level, counteracts neuroinflammatory markers and rescues cognitive deficits in a murine model of Alzheimer's disease.Blood amyloid beta levels in healthy, mild cognitive impairment and Alzheimer's disease individuals: replication of diastolic blood pressure correlations and analysis of critical covariates.Metabolic Dysfunction of Astrocyte: An Initiating Factor in Beta-amyloid Pathology?Both creatine and its product phosphocreatine reduce oxidative stress and afford neuroprotection in an in vitro Parkinson's model.The anti-AD cookbook: a new recipe.
P2860
High-energy compounds promote physiological processing of Alzheimer's amyloid-β precursor protein and boost cell survival in culture.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh-hant
name
High-energy compounds promote ...... oost cell survival in culture.
@en
High-energy compounds promote ...... oost cell survival in culture.
@nl
type
label
High-energy compounds promote ...... oost cell survival in culture.
@en
High-energy compounds promote ...... oost cell survival in culture.
@nl
prefLabel
High-energy compounds promote ...... oost cell survival in culture.
@en
High-energy compounds promote ...... oost cell survival in culture.
@nl
P2093
P2860
P1476
High-energy compounds promote ...... oost cell survival in culture.
@en
P2093
Huey T Nguyen
Olga Markov
P2860
P304
P356
10.1111/J.1471-4159.2012.07923.X
P407
P577
2012-09-21T00:00:00Z