Comparative study of microglia activation induced by amyloid-beta and prion peptides: role in neurodegeneration.
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Prion peptide uptake in microglial cells--the effect of naturally occurring autoantibodies against prion proteinExpression of a Cu,Zn superoxide dismutase typical for familial amyotrophic lateral sclerosis increases the vulnerability of neuroblastoma cells to infectious injuryMicroglia in infectious diseases of the central nervous system.CD36 participates in PrP(106-126)-induced activation of microglia.The NALP3 inflammasome is involved in neurotoxic prion peptide-induced microglial activationIsolated amyloid-β(1-42) protofibrils, but not isolated fibrils, are robust stimulators of microglia.Microglia, neuroinflammation, and beta-amyloid protein in Alzheimer's disease.Different Molecular Mechanisms Mediate Direct or Glia-Dependent Prion Protein Fragment 90-231 Neurotoxic Effects in Cerebellar Granule Neurons.Amyloid-β Activates Microglia and Regulates Protein Expression in a Manner Similar to Prions.Toll-like receptor 2 deficiency shifts PrP106-126-induced microglial activation from a neurotoxic to a neuroprotective phenotype.Fibrillar β-amyloid 1-42 alters cytokine secretion, cholinergic signalling and neuronal differentiation.Combined Creutzfeldt-Jakob-Jacob/ Alzheimer's Disease Cases are Important in Search for Microbes in Alzheimer's Disease.14-3-3ε marks the amyloid-stimulated microglia long-term activation.Effects of immunomodulatory substances on phagocytosis of abeta(1-42) by human microglia.Suppressed microglial E prostanoid receptor 1 signaling selectively reduces tumor necrosis factor alpha and interleukin 6 secretion from toll-like receptor 3 activation.The effect of intrahippocampal beta amyloid (1-42) peptide injection on oxidant and antioxidant status in rat brain.Interactions of the prion peptide (PrP 106-126) with brain capillary endothelial cells: coordinated cell killing and remodeling of intercellular junctions.Effect of PrP105-132 on the secretion of interleukin-6 and interleukin-8 from microglial cells in vitro.Adenosine A2A receptors modulate glutamate uptake in cultured astrocytes and gliosomes.Antibody-mediated inhibition of integrin α5β1 blocks neurotoxic prion peptide PrP106-126-induced activation of BV2 microglia
P2860
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P2860
Comparative study of microglia activation induced by amyloid-beta and prion peptides: role in neurodegeneration.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh
2006年學術文章
@zh-hant
name
Comparative study of microglia ...... es: role in neurodegeneration.
@en
Comparative study of microglia ...... es: role in neurodegeneration.
@nl
type
label
Comparative study of microglia ...... es: role in neurodegeneration.
@en
Comparative study of microglia ...... es: role in neurodegeneration.
@nl
prefLabel
Comparative study of microglia ...... es: role in neurodegeneration.
@en
Comparative study of microglia ...... es: role in neurodegeneration.
@nl
P356
P1476
Comparative study of microglia ...... es: role in neurodegeneration.
@en
P2093
Pedro Garção
P304
P356
10.1002/JNR.20870
P577
2006-07-01T00:00:00Z