Ionic mechanisms of spontaneous GABAergic events in rat hippocampal slices exposed to 4-aminopyridine.
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Ion dynamics during seizuresCarbonic anhydrase I, II, and VI, blood plasma, erythrocyte and saliva zinc and copper increase after repetitive transcranial magnetic stimulation.Selective activation of parvalbumin- or somatostatin-expressing interneurons triggers epileptic seizurelike activity in mouse medial entorhinal cortex.Hippocampal neuron firing and local field potentials in the in vitro 4-aminopyridine epilepsy model.Synchronous GABA-receptor-dependent potentials in limbic areas of the in-vitro isolated adult guinea pig brain.Hilar somatostatin interneurons contribute to synchronized GABA activity in an in vitro epilepsy model.Inhibition of carbonic anhydrase augments GABAA receptor-mediated analgesia via a spinal mechanism of action.Spatial and temporal dynamics in the ionic driving force for GABA(A) receptors.GABAergic synchronization in the limbic system and its role in the generation of epileptiform activity.Optogenetic activation of VGLUT2-expressing excitatory neurons blocks epileptic seizure-like activity in the mouse entorhinal cortexShort-term ionic plasticity at GABAergic synapses.Cl-out is a novel cooperative optogenetic tool for extruding chloride from neurons.Neurosteroids modulate epileptiform activity and associated high-frequency oscillations in the piriform cortex.The K+-Cl cotransporter KCC2 promotes GABAergic excitation in the mature rat hippocampus.Synchronization of GABAergic inputs to CA3 pyramidal cells precedes seizure-like event onset in juvenile rat hippocampal slices.Synaptic and nonsynaptic contributions to giant ipsps and ectopic spikes induced by 4-aminopyridine in the hippocampus in vitro.Dynamic modulation of excitation and inhibition during stimulation at gamma and beta frequencies in the CA1 hippocampal region.GABA application to hippocampal CA3 or CA1 stratum lacunosum-moleculare excites an interneuron network.Rat subicular networks gate hippocampal output activity in an in vitro model of limbic seizures.Cl- accumulation does not account for the depolarizing phase of the synaptic GABA response in hippocampal pyramidal cells.Transition to seizures in the isolated immature mouse hippocampus: a switch from dominant phasic inhibition to dominant phasic excitation.Optogenetic dissection of roles of specific cortical interneuron subtypes in GABAergic network synchronization.Propagation of postsynaptic currents and potentials via gap junctions in GABAergic networks of the rat hippocampus.
P2860
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P2860
Ionic mechanisms of spontaneous GABAergic events in rat hippocampal slices exposed to 4-aminopyridine.
description
1997 nî lūn-bûn
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name
Ionic mechanisms of spontaneou ...... es exposed to 4-aminopyridine.
@en
Ionic mechanisms of spontaneou ...... es exposed to 4-aminopyridine.
@nl
type
label
Ionic mechanisms of spontaneou ...... es exposed to 4-aminopyridine.
@en
Ionic mechanisms of spontaneou ...... es exposed to 4-aminopyridine.
@nl
prefLabel
Ionic mechanisms of spontaneou ...... es exposed to 4-aminopyridine.
@en
Ionic mechanisms of spontaneou ...... es exposed to 4-aminopyridine.
@nl
P1476
Ionic mechanisms of spontaneou ...... es exposed to 4-aminopyridine.
@en
P304
P356
10.1152/JN.1997.78.5.2582
P407
P577
1997-11-01T00:00:00Z