Decreased hemispheric Aquaporin-4 is linked to evolving brain edema following controlled cortical impact injury in rats.
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Role of Glia in Memory Deficits Following Traumatic Brain Injury: Biomarkers of Glia DysfunctionDynamic regulation of aquaporin-4 water channels in neurological disordersActivation of P2X7 promotes cerebral edema and neurological injury after traumatic brain injury in miceRegional expression of aquaporin 1, 4, and 9 in the brain during pregnancyCorrespondence of AQP4 expression and hypoxic-ischaemic brain oedema monitored by magnetic resonance imaging in the immature and juvenile rat.Efficacy and toxicity of the antisense oligonucleotide aprinocarsen directed against protein kinase C-alpha delivered as a 21-day continuous intravenous infusion in patients with recurrent high-grade astrocytomas.The protein kinase C activator phorbol myristate acetate decreases brain edema by aquaporin 4 downregulation after middle cerebral artery occlusion in the ratEffect of secondary insults upon aquaporin-4 water channels following experimental cortical contusion in rats.Gene expression profiling of astrocytes from hyperammonemic mice reveals altered pathways for water and potassium homeostasis in vivo.Curcumin attenuates cerebral edema following traumatic brain injury in mice: a possible role for aquaporin-4?Differential aquaporin 4 expression during edema build-up and resolution phases of brain inflammation.Aquaporin and vascular diseases.Experimental traumatic brain injuryThe apparent diffusion coefficient does not reflect cytotoxic edema on the uninjured side after traumatic brain injuryCritical role of NADPH oxidase in neuronal oxidative damage and microglia activation following traumatic brain injury.Delayed increase of astrocytic aquaporin 4 after juvenile traumatic brain injury: possible role in edema resolution?An alpha-syntrophin-dependent pool of AQP4 in astroglial end-feet confers bidirectional water flow between blood and brainAquaporin expression in the brains of patients with or without cerebral amyloid angiopathyDifferent expressions of AQP1, AQP4, eNOS, and VEGF proteins in ischemic versus non-ischemic cerebropathy in rats: potential roles of AQP1 and eNOS in hydrocephalic and vasogenic edema formation.A neurovascular perspective for long-term changes after brain trauma.Mildly Reduced Brain Swelling and Improved Neurological Outcome in Aquaporin-4 Knockout Mice following Controlled Cortical Impact Brain Injury.Lost Polarization of Aquaporin4 and Dystroglycan in the Core Lesion after Traumatic Brain Injury Suggests Functional Divergence in Evolution.Glial cell aquaporin-4 overexpression in transgenic mice accelerates cytotoxic brain swelling.'Hit & Run' model of closed-skull traumatic brain injury (TBI) reveals complex patterns of post-traumatic AQP4 dysregulationAquaporin and brain diseasesTherapeutic targeting of astrocytes after traumatic brain injury.Pathogenesis of brain edema and investigation into anti-edema drugs.Aquaporins in Nervous System.Endothelins decrease the expression of aquaporins and plasma membrane water permeability in cultured rat astrocytes.Potential utility of aquaporin modulators for therapy of brain disorders.Rapamycin alleviates brain edema after focal cerebral ischemia reperfusion in rats.Hyperosmolar mannitol simulates expression of aquaporins 4 and 9 through a p38 mitogen-activated protein kinase-dependent pathway in rat astrocytes.The role of vasopressin V1A receptors in cytotoxic brain edema formation following brain injury.Cerebral cortical aquaporin-4 expression in brain edema following cardiac arrest in rats.Blood-brain barrier disruption highly induces aquaporin-4 mRNA and protein in perivascular and parenchymal astrocytes: protective effect by estradiol treatment in ovariectomized animals.Effect of ultrasound treatment on brain edema in a traumatic brain injury model with the weight drop method.Erythropoietin Attenuates the Brain Edema Response after Experimental Traumatic Brain Injury.AQP5 is differentially regulated in astrocytes during metabolic and traumatic injuries.Sulfonylurea Receptor-1: A Novel Biomarker for Cerebral Edema in Severe Traumatic Brain Injury.Glibenclamide produces region-dependent effects on cerebral edema in a combined injury model of traumatic brain injury and hemorrhagic shock in mice.
P2860
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P2860
Decreased hemispheric Aquaporin-4 is linked to evolving brain edema following controlled cortical impact injury in rats.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Decreased hemispheric Aquapori ...... ortical impact injury in rats.
@en
Decreased hemispheric Aquapori ...... ortical impact injury in rats.
@nl
type
label
Decreased hemispheric Aquapori ...... ortical impact injury in rats.
@en
Decreased hemispheric Aquapori ...... ortical impact injury in rats.
@nl
prefLabel
Decreased hemispheric Aquapori ...... ortical impact injury in rats.
@en
Decreased hemispheric Aquapori ...... ortical impact injury in rats.
@nl
P2093
P1433
P1476
Decreased hemispheric Aquapori ...... cortical impact injury in rats
@en
P2093
Andreas W Unterberg
John F Stover
Karl L Kiening
Stefan Schreiber
Ulrich W Thomale
P304
P356
10.1016/S0304-3940(02)00180-5
P407
P577
2002-05-01T00:00:00Z