Relationship between glial potassium regulation and axon excitability: a role for glial Kir4.1 channels.
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Autoantibodies to Potassium Channel KIR4.1 in Multiple SclerosisAstrocytes differentially respond to inflammatory autoimmune insults and imbalances of neural activity.Astrocytic and neuronal accumulation of elevated extracellular K(+) with a 2/3 K(+)/Na(+) flux ratio-consequences for energy metabolism, osmolarity and higher brain function.Aquaporin-4-dependent K(+) and water transport modeled in brain extracellular space following neuroexcitationDiffusion fMRI detects white-matter dysfunction in mice with acute optic neuritisPhysiological bases of the K+ and the glutamate/GABA hypotheses of epilepsy.Sertraline Induces Toxicity and Behavioral Alterations in Planarians.Antagonists of the Vasopressin V1 Receptor and of the β(1)-Adrenoceptor Inhibit Cytotoxic Brain Edema in Stroke by Effects on Astrocytes - but the Mechanisms Differ.Contributions of the Na⁺/K⁺-ATPase, NKCC1, and Kir4.1 to hippocampal K⁺ clearance and volume responses.Regulation of neurovascular coupling in autoimmunity to water and ion channelsElectrodiffusive model for astrocytic and neuronal ion concentration dynamics.The neuroglial potassium cycle during neurotransmission: role of Kir4.1 channelsVoltage-dependent K+ currents contribute to heterogeneity of olfactory ensheathing cellsLoss of Local Astrocyte Support Disrupts Action Potential Propagation and Glutamate Release Synchrony from Unmyelinated Hippocampal Axon Terminals In VitroThe computational power of astrocyte mediated synaptic plasticity.Astrocytes modulate neural network activity by Ca²+-dependent uptake of extracellular K+.Vestibular role of KCNQ4 and KCNQ5 K+ channels revealed by mouse models.Expression of Kir4.1 and Kir5.1 inwardly rectifying potassium channels in oligodendrocytes, the myelinating cells of the CNS.How do astrocytes shape synaptic transmission? Insights from electrophysiology.Astrocytic glycogenolysis: mechanisms and functions.Roles of astrocytic Na(+),K(+)-ATPase and glycogenolysis for K(+) homeostasis in mammalian brain.Role of the Astrocytic Na(+), K(+)-ATPase in K(+) Homeostasis in Brain: K(+) Uptake, Signaling Pathways and Substrate Utilization.The role of glial-specific Kir4.1 in normal and pathological states of the CNS.Multifactorial Effects on Different Types of Brain Cells Contribute to Ammonia Toxicity.TASK-1 channels in oligodendrocytes: a role in ischemia mediated disruptionAstroglial calcium signaling displays short-term plasticity and adjusts synaptic efficacy.Age-Related Changes in Axonal and Mitochondrial Ultrastructure and Function in White Matter.Kir4.1-mediated spatial buffering of K(+): experimental challenges in determination of its temporal and quantitative contribution to K(+) clearance in the brain.Integration between Glycolysis and Glutamate-Glutamine Cycle Flux May Explain Preferential Glycolytic Increase during Brain Activation, Requiring Glutamate.Can a Selective Serotonin Reuptake Inhibitor Act as a Glutamatergic Modulator?Sequential Astrocytic 5-HT2B Receptor Stimulation, [Ca(2+)]i Regulation, Glycogenolysis, Glutamate Synthesis, and K(+) Homeostasis are Similar but Not Identical in Learning and Mood Regulation.When can AQP4 assist transporter-mediated K⁺ uptake?Book review: “Glial physiology and pathophysiology” by Alexei Verkhratsky and Arthur Butt, Wiley-Blackwell, 2013.Cell-specific mRNA alterations in Na+, K+-ATPase α and β isoforms and FXYD in mice treated chronically with carbamazepine, an anti-bipolar drug.Requirement of glycogenolysis for uptake of increased extracellular K+ in astrocytes: potential implications for K+ homeostasis and glycogen usage in brain.Severe Convulsions and Dysmyelination in Both Jimpy and Cx32/47 -/- Mice may Associate Astrocytic L-Channel Function with Myelination and Oligodendrocytic Connexins with Internodal Kv Channels.Time-course of glial changes in the hyperhomocysteinemia model of vascular cognitive impairment and dementia (VCID).Astrocytic Kir4.1 channels and gap junctions account for spontaneous epileptic seizure.Oligodendrocytes control potassium accumulation in white matter and seizure susceptibility.Oligodendrocyte-encoded Kir4.1 function is required for axonal integrity
P2860
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P2860
Relationship between glial potassium regulation and axon excitability: a role for glial Kir4.1 channels.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh-hant
name
Relationship between glial pot ...... ole for glial Kir4.1 channels.
@en
Relationship between glial pot ...... ole for glial Kir4.1 channels.
@nl
type
label
Relationship between glial pot ...... ole for glial Kir4.1 channels.
@en
Relationship between glial pot ...... ole for glial Kir4.1 channels.
@nl
prefLabel
Relationship between glial pot ...... ole for glial Kir4.1 channels.
@en
Relationship between glial pot ...... ole for glial Kir4.1 channels.
@nl
P2860
P356
P1433
P1476
Relationship between glial pot ...... ole for glial Kir4.1 channels.
@en
P2093
Virginia Bay
P2860
P304
P356
10.1002/GLIA.22299
P50
P577
2012-01-30T00:00:00Z