Cortical focus drives widespread corticothalamic networks during spontaneous absence seizures in rats.
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A brief history on the oscillating roles of thalamus and cortex in absence seizuresUncovering the neurobehavioural comorbidities of epilepsy over the lifespanNew vistas on astroglia in convulsive and non-convulsive epilepsy highlight novel astrocytic targets for treatmentA computational study of stimulus driven epileptic seizure abatement.CaV3.2 calcium channels control NMDA receptor-mediated transmission: a new mechanism for absence epilepsy.Advantages of repeated low dose against single high dose of kainate in C57BL/6J mouse model of status epilepticus: behavioral and electroencephalographic studiesPresence of a Chaotic Region at the Sleep-Wake Transition in a Simplified Thalamocortical Circuit ModelExcitatory amplification through divergent-convergent circuits: the role of the midline thalamus in limbic seizures.Neuronal circuits in epilepsy: do they matter?Temporal lobe epilepsy: where do the seizures really begin?Reduced cortical inhibition in a mouse model of familial childhood absence epilepsy.Absence seizures in C3H/HeJ and knockout mice caused by mutation of the AMPA receptor subunit Gria4Clinical use of ictal SPECT in secondarily generalized tonic-clonic seizuresCortical and subcortical networks in human secondarily generalized tonic-clonic seizuresDeletion of phospholipase C beta4 in thalamocortical relay nucleus leads to absence seizures.Focal BOLD fMRI changes in bicuculline-induced tonic-clonic seizures in the rat.Dynamic time course of typical childhood absence seizures: EEG, behavior, and functional magnetic resonance imaging.Focal structural changes and cognitive dysfunction in juvenile myoclonic epilepsy.A new mode of corticothalamic transmission revealed in the Gria4(-/-) model of absence epilepsy.Altered functional connectivity within and between brain modules in absence epilepsy: a resting-state functional magnetic resonance imaging study.CaV 2.1 ablation in cortical interneurons selectively impairs fast-spiking basket cells and causes generalized seizures.Workshop on Neurobiology of Epilepsy appraisal: new systemic imaging technologies to study the brain in experimental models of epilepsySpatiotemporal dynamics of optogenetically induced and spontaneous seizure transitions in primary generalized epilepsy.Reduction of thalamic and cortical Ih by deletion of TRIP8b produces a mouse model of human absence epilepsy.Insights into the mechanisms of absence seizure generation provided by EEG with functional MRI.The enhanced information flow from visual cortex to frontal area facilitates SSVEP response: evidence from model-driven and data-driven causality analysis.Consciousness and epilepsy: why are patients with absence seizures absent?Persistence of cortical sensory processing during absence seizures in human and an animal model: evidence from EEG and intracellular recordings.Identifying neural drivers with functional MRI: an electrophysiological validation.Causal hierarchy within the thalamo-cortical network in spike and wave discharges.Reduced local input to fast-spiking interneurons in the somatosensory cortex in the GABAA γ2 R43Q mouse model of absence epilepsy.Cross-approximate entropy of cortical local field potentials quantifies effects of anesthesia--a pilot study in rats.Neuroimaging biomarkers of epileptogenesis.Concepts of connectivity and human epileptic activityCombined Effects of Feedforward Inhibition and Excitation in Thalamocortical Circuit on the Transitions of Epileptic SeizuresAberrant GABA(A) receptor-mediated inhibition in cortico-thalamic networks of succinic semialdehyde dehydrogenase deficient mice.Transition to absence seizures and the role of GABA(A) receptorsWhere fMRI and electrophysiology agree to disagree: corticothalamic and striatal activity patterns in the WAG/Rij rat.Interactions between membrane conductances underlying thalamocortical slow-wave oscillationsLocal changes in neocortical circuit dynamics coincide with the spread of seizures to thalamus in a model of epilepsy.
P2860
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P2860
Cortical focus drives widespread corticothalamic networks during spontaneous absence seizures in rats.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Cortical focus drives widespre ...... eous absence seizures in rats.
@en
Cortical focus drives widespre ...... eous absence seizures in rats.
@nl
type
label
Cortical focus drives widespre ...... eous absence seizures in rats.
@en
Cortical focus drives widespre ...... eous absence seizures in rats.
@nl
prefLabel
Cortical focus drives widespre ...... eous absence seizures in rats.
@en
Cortical focus drives widespre ...... eous absence seizures in rats.
@nl
P2093
P1476
Cortical focus drives widespre ...... neous absence seizures in rats
@en
P2093
Anton M L Coenen
Egidius L J M Van Luijtelaar
Fernando H Lopes da Silva
Hanneke K M Meeren
Jan Pieter M Pijn
P304
P356
10.1523/JNEUROSCI.22-04-01480.2002
P407
P577
2002-02-01T00:00:00Z